「cancer」の共起表現一覧(1語右が「cells」)
該当件数 : 62件
ts participate in the selective destruction of | cancer cells, or inhibit the growth and proliferation |
Removing | cancer cells from the abdominal cavity reduces the tu |
he inhibition of the proliferation of prostate | cancer cells, have also been investigated. |
ltimately leads to apoptosis in WT human colon | cancer cells but not in human colon cancer cells lack |
Cancer cells frequently have aneuploidy of chromosome | |
ntified a way to exploit genetic weaknesses in | cancer cells including mutated BRCA 1 or BRCA2, leadi |
in fusion transcripts are commonly produced by | cancer cells, and detection of fusion transcripts is |
Many types of | cancer cells carry EpCAM (epithelial cell adhesion mo |
aller extent, testosterone, stimulate prostate | cancer cells to grow. |
CD47 is used by bladder | cancer cells to hide from normal scavenging by macrop |
egulation of estrogen receptor negative breast | cancer cells transfected with cDNA's for estrogen rec |
It acts to freeze | cancer cells in phase G1 of the cell cycle, preventin |
The enforced expression causes the growth of | cancer cells to slow down. |
Prototypes that separate | cancer cells from healthy cells have been made. |
In | cancer cells, EP300 mutations prevent the gene from p |
N-Cadherin is commonly found in | cancer cells and provides a mechanism for transendoth |
e high levels of SP2 protein that are found in | cancer cells would be neutralized. |
ic acid was found to show cytotoxicity against | cancer cells, without harming healthy cells. |
It slows or stops the growth of | cancer cells in the body. |
Importantly, vitamin D treatment of colon | cancer cells, Caco-2, also increases expression of TR |
so able to kill human leukemic cells and colon | cancer cells by apoptosis. |
t cancers as protective factors for the breast | cancer cells makes them important targets for structu |
ve pro-adhesive and chemotactic activities for | cancer cells, suggesting that this molecule may have |
n coded with the MDR1 gene manifests itself in | cancer cells, it discharges much of the antineoplasti |
serve as adhesion molecule for lung metastatic | cancer cells, mediating vascular arrest and colonizat |
Cancer cells generally divide more often than healthy | |
Splice variants of CD44 on colon | cancer cells display sialofucosylated HCELL glycoform |
It is found on the surface of many types of | cancer cells and shed into the blood stream. |
Transfection of breast | cancer cells with ferroportin significantly reduces t |
found that the overexpression of H19 in breast | cancer cells promotes proliferation. |
oproteinases, a function important to invading | cancer cells within bone. |
In human | cancer cells DNMT1 is responsible for both de novo an |
type in estrogen receptor (ER)-positive breast | cancer cells with deficient intercellular adhesion. |
stically act with sirolimus to kill pancreatic | cancer cells in the laboratory in a Mayo Clinic study |
an antibody-drug conjugate (ADC) that targets | cancer cells expressing transmembrane glycoprotein NM |
The antibody binds to GPNMB on the | cancer cells, the ADC is internalised, the linkage is |
It is highly potent agent, capable of damage | cancer cells in very low concentrations, and retains |
s (an immortal cell line derived from cervical | cancer cells), but was later found to be ubiquitously |
ignaling and are frequently seen in metastatic | cancer cells that are invading surrounding tissues. |
to mesothelin (a protein that is made by some | cancer cells) and stops the cells from dividing. |
p53-deficient human gastric | cancer cells, restoration of functional miR-34 inhibi |
idar inhibits this P-glycoprotein, causing the | cancer cells to lose their medicine tolerance, and ma |
similarities in the gene pathways activated in | cancer cells, and those activated in cells involved i |
genetically-enhanced virus based on Ebola and | cancer cells, which they plan to use to wipe out much |
hyllotoxin derivatives and slows the growth of | cancer cells in the body. |
proven ability to inhibit energy metabolism in | cancer cells, and to enhance the activity of anticanc |
ant role in the immune system's recognition of | cancer cells, so indiscriminate blocking of IgE / rec |
onsible for the invasive properties of hypoxic | cancer cells through focal adhesion kinase activity a |
CBD reduces growth of aggressive human breast | cancer cells in vitro and reduces their invasiveness. |
ction is that the compound binds to the DNA of | cancer cells rendering them incapable of dividing. |
fect all rapidly dividing cell types including | cancer cells, but also those of intestinal epithelium |
es cytochrome c release in the mitochondria of | cancer cells leading to cell death, but does not harm |
cin is able to trigger apoptosis in human lung | cancer cells as well. |
increased proliferation and migration of colon | cancer cells, which might encourage cancer cells to m |
ed by microwave irradiation to damage and kill | cancer cells or to make cancer cells more sensitive t |
c gene variations, or molecular signatures, in | cancer cells and use that knowledge to accelerate the |
d chitosan, has been shown to transfect breast | cancer cells; with increased degree of trimethylation |
It works by damaging DNA, primarily in | cancer cells that preferentially take up the uracil d |
f killing GPNMB expressing melanoma and breast | cancer cells in vitro and inducing partial or complet |
of PCGEM1 were found to be higher in prostate | cancer cells in African-American men than in Caucasia |
may have an anti-proliferative effect on colon | cancer cells and an anti-diabetic effect by means of |
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