「insulin」の共起表現(1語左で並び替え)2ページ目

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共起表現

「insulin」の共起表現一覧(1語左で並び替え)2ページ目

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returning to Toronto to begin his research on	insulin in the spring of 1921.
iling health; a diabetic, she is dependent on	insulin to control her condition but has to obtain su
d that American men with abdominal obesity or	insulin resistance (a precursor to diabetes) were mor
ic hypoglycaemia, induced by an over-generous	insulin and had not neutralised its effects with food
ino acid in polypeptide chains, in particular	insulin.
adhesion, b cell receptor signaling pathway,	insulin signaling pathway, melanogenesis, alzheimer's
It seems also to have a role in peripheral	insulin resistance.
reatment of obesity, for enhancing peripheral	insulin sensitivity in patients with type-2 diabetes
As a phosphoprotein phosphatase,	insulin dephosphorylates the enzyme, thus activating
normalising plasma	insulin levels
Hx are potent	insulin secretagogues.
are physiologically important in potentiating	insulin release.
Collip's task was to prepare	insulin in a more pure, usable form than Banting and
nto almost any organ-and coax them to produce	insulin in diabetics.
e able to sense the glucose level and produce	insulin, the amount of insulin secreted is usually th
The pancreas must produce	insulin for this medication to work.
The group succeeded in producing	insulin in 1978, and in 1979, Riggs received the Juve
the body attacks the cells producing	insulin, the chemical that allows the metabolizing of
promotes	insulin sensitivity.
nage blood sugar: on condition that providing	insulin stimulating meds or meds that sensitizes the
MUC1, Mammalian target of rapamycin, RASGRP3,	Insulin receptor, C1QBP, PLD2 and SHC1.
ulating neurotransmitter release, heart rate,	insulin secretion, neuronal excitability, epithelial
can specify the frequency of their real-life	insulin injections.
from the Surviving Sepsis Campaign recommend	insulin therapy in critically ill patients.
th PCOS (polycystic ovary syndrome) to reduce	insulin problems and possibly improve fertility.
was hypothesized that the grapefruit reduced	insulin levels, encouraging fat loss.
Hyperinsulinism due to reduced	insulin sensitivity is usually asymptomatic.
lowing slightly more flexibility than regular	insulin, which requires a longer waiting period befor
m of action by which these complexes regulate	insulin signaling pathways is unclear.
plex, bis(allixinato)zinc(II), shows the same	insulin mimetic effects.
an overeaters' increased tendency to secrete	insulin at the sight and smell of food, though medica
sugar by stimulating the pancreas to secrete	insulin and helping the body use insulin efficiently.
warded the Nobel Prize in 1958 for sequencing	insulin.)
ns with GAD65 has been shown to preserve some	insulin production for 30 months in humans with type
r form of the disease, in which there is some	insulin resistance but normal growth and subcutaneous
-glucose pentaacetate, was found to stimulate	insulin release, and might therefore be of therapeuti
GIP stimulates	insulin release when glucose concentration is elevate
c islets causing calcium entry and subsequent	insulin release.
ease and is linked to the metabolic syndrome (	insulin insensitivity).
de production, nerve growth factor synthesis,	insulin resistance, loss of mean body mass, and IL-8
tile chemistry were the first who synthesised	Insulin.
n credited with being the first to synthesize	insulin while leading a team at the University of Pit
His results synthesizing	insulin were achieved almost simultaneously with that
roducts and/or processes, including synthetic	insulin, growth hormone, and TPA, often beating out b
pupil who will go as far as refusing to take	insulin so that she can lose weight, to be "Rochdales
e became ill in mid-1944, but refused to take	insulin due to his devout Christian Scientist beliefs
Studies have shown that patients taking	insulin degludec needed to take significantly smaller
The statement that '	insulin has been strongly preserved' is vague, as it
nts”, Somogyi first presented his theory that	insulin treatment in itself might cause unstable diab
cogen storage levels, and this indicates that	insulin activates glycogen synthase by inhibiting its
treatment for depression in the same way that	insulin shock therapy was for schizophrenia but I can
Sanger's initial results suggested that	insulin was a smaller molecule than previously estima
genes such as those for H-ras, c-myb, and the	insulin receptor,, and genes encoding housekeeping en
cell surface, the topological mapping of the	insulin signal transduction pathway, the creation of
he kinase domains of the EGFR and that of the	insulin receptor.
the sulfonylureas group gained 3 kg, and the	insulin group, 6 kg.
This can change the potency of the	insulin, and it would no longer be as effective.
sphorylation of IRS-1, thereby inhibiting the	insulin signalling cascade.
for example on the peripheral cells (e.g. the	insulin releasing cells of the pancreas) and on the c
here is a 60% homology between IGF-1R and the	insulin receptor.
The main activity of activation of the	insulin receptor is inducing glucose uptake.
Glycogen synthesis is also stimulated by the	insulin receptor via IRS-1.
he relaxin-like peptide family belongs in the	insulin superfamily and consists of 7 peptides of hig
vival of progenitor myeloid cells through the	insulin receptor substrate-2/PI 3-kinase/AKT pathway.
1967 in connection with the discovery of the	insulin biosynthesis.
so appears to have a beneficial effect on the	insulin resistance featured by the metabolic syndrome
mple of tyrosine kinase receptors include the	insulin receptor, the IGF-1 receptor, the MuSK protei
isease, having only one normal allele for the	insulin receptor) will not be affected.
cell cycle progression due to its role in the	insulin / TOR / S6K signaling pathway.
former student of his father, on cloning the	insulin gene from carp.
oblastoma, which is an abnormal growth of the	insulin secreting cells in the pancreas.
region (ILPR) is a regulatory sequence on the	insulin gene starting at position -363 upstream from
The	Insulin Signaling Pathway
In molecular biology, the	insulin receptor is a transmembrane receptor that is
A3 is a regulatory sequence for the	insulin gene
lin is the primary translation product of the	insulin gene.
C1 is a regulatory sequence for the	insulin gene
A2 is a regulatory sequence for the	insulin gene
Z is a regulatory sequence for the	insulin gene
G1 is a regulatory sequence for the	insulin gene
DBP binds to a upstream promoter in the	insulin gene.
A5 is a regulatory sequence for the	insulin gene
role in signal transduction downstream of the	insulin receptor.
periments had included patients' having their	insulin withheld for two days, during which time they
rodents, while also remembering to take their	insulin and check their blood glucose.
This	insulin secretion subsides as blood glucose concentra
sors, such as the conversion of proinsulin to	insulin intermediates.
hormone can reduce a person's sensitivity to	insulin.
the major tissues in the body that respond to	insulin.
that codes for a zinc transporter related to	insulin secretion in humans.
It is assumed that the ability to respond to	insulin induced hypoglycemia translates into appropri
disulfide bridges, and it appears related to	insulin.
: those tending toward reduced sensitivity to	insulin and high blood glucose levels (hyperglycemia)
by removal of the signal peptide and then to	insulin by removal of the connecting peptide (C-pepti
rds a similar level of blood sugar control to	insulin and sulfonylureas, it appears to decrease mor
uding regulation of vascular and airway tone,	insulin secretion, and peristalsis.
include glutathione-insulin transhydrogenase,	insulin reductase, reductase, protein disulfide (glut
ows continued signal output (e.g., to trigger	insulin release) amid significant amounts of its prod
dren, and Barbara is a diabetic who needs two	insulin shots per day.
e reason, preproinsulin is rarely used unlike	insulin, the mature product, and proinsulin, a stable
It is claimed to help build up	insulin in the human body and is sometimes referred t
in metabolic control in many animals, whether	insulin per se is important in ALL animals is, I beli
effect and may be beneficial for people with	insulin resistance.
pression of the receptors are correlated with	insulin levels, as well as reduced in mouse models of
Not to be confused with	insulin.
eam by the β-cells of the pancreas along with	insulin, after a meal.
n the Insulin-Receptor Gene in a Patient with	Insulin Resistance, Acanthosis Nigricans, and the Pol
all mortality by about 30% when compared with	insulin and sulfonylureas (glibenclamide and chlorpro
Both types are also associate with	insulin resistance.
delay progress to diabetes, and in those with	insulin resistance it slowed progress in the HOMA sev
for the murder of an 80-year-old patient with	insulin, has been identified as a possible inspiratio

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「insulin」の共起表現一覧(1語左で並び替え)2ページ目