「ACETYLCHOLINE」の共起表現(1語右で並び替え)

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共起表現

「ACETYLCHOLINE」の共起表現一覧(1語右で並び替え)

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hrocyte cholinesterase, or (most formally)	acetylcholine acetylhydrolase, found primarily in the bl
cholinesterase functions to deactivate the	acetylcholine almost immediately by breaking it down.
ich has a short action time, is similar to	acetylcholine and acts as a partial agonist of the nicot
It also increases release of	acetylcholine and noradrenaline, and improves memory ret
They are stimulated by muscarine and	acetylcholine, and blocked by atropine.
The structure of the binding site for the	acetylcholine and nicotine was located at the interface
hances the potassium-stimulated release of	acetylcholine and glutamate.
rtant central neurotransmitters, including	acetylcholine and glutamate have been reported.
nzyme choline acetyltransferase to produce	acetylcholine and a coenzyme a byproduct.
his work on neuromodulators, particularly	acetylcholine, and for his computational modelling work
e symptoms of schizophrenia, and increases	acetylcholine and glutamate levels in the prefrontal cor
ns that release the transmitter substances	acetylcholine and serotonin.
ts primarily as a selective α3β4 nicotinic	acetylcholine antagonist, and is even more selective tha
It is therefore classified as an indirect	acetylcholine antagonist.
o found to act as selective α3β4 nicotinic	acetylcholine antagonists, with little or no effect on N
	acetylcholine antagonists.
A receptor is cholinergic if it uses	acetylcholine as its neurotransmitter.
For his study of	acetylcholine as agent in the chemical transmission of n
erase by phosphorylation, so the action of	acetylcholine becomes prolonged, pralidoxime (2-PAM) is
known as muscarinic receptor antagonists -	acetylcholine being the neurotransmitter of the parasymp
Unlike	acetylcholine, bethanechol is not hydrolyzed by cholines
N) to respond to a neurotransmitter called	acetylcholine; BFCN are important for the processes of l
After	acetylcholine binding, the receptor undergoes an extensi
With the enzyme inhibited,	acetylcholine builds up in the synapse and continues to
All three ACh-inhibiting (	Acetylcholine) chemicals can be found in the leaves, ste
AChE is an enzyme that removes	acetylcholine from the synapse after it creates the requ
g further effects to the normal release of	acetylcholine from the presynaptic terminal, and therefo
annel superfamily is composed of nicotinic	acetylcholine, GABAA, GABAA-ρ, glycine and 5-HT3 recepto
the rate limiting step in the synthesis of	acetylcholine; hence, hemicholinium-3 decreases the synt
ssible neurotransmitter substances such as	acetylcholine, histamine, substance P, and serotonin.
	Acetylcholine hyperpolarizes the sinoatrial node which i
Intake of	acetylcholine in axoplasm is prevented and the presynapt
inhibitor which prevents the breakdown of	acetylcholine in the synapse.
, the acetylcholinesterase breaks down the	acetylcholine in the synaptic cleft in order to allow th
nsmission by interfering with synthesis of	acetylcholine in nerve endings.
rgic drug designed to reduce the levels of	acetylcholine in the treatment of Parkinson's disease.
It stimulates release of dopamine and	acetylcholine in the brain in both rodent and primate mo
AMPA-mediated release of noradrenaline and	acetylcholine in the hippocampus and frontal cortex of t
r asystole during Stokes-Adams attacks and	acetylcholine injections.
nism of breaking down the neurotransmitter	acetylcholine into its constituents, acetate and choline
yze the hydrolysis of the neurotransmitter	acetylcholine into choline and acetic acid, a reaction n
	Acetylcholine is synthesized from choline and a donated
	Acetylcholine is associated with attention, concentratio
If it gets inhibited,	acetylcholine is not removed after the stimulation and m
Once this receptor binds	acetylcholine, it undergoes an extensive change in confo
ed in animals, and found to increase brain	acetylcholine levels and produce nootropic effects, as w
nown to be a partial agonist of muscarinic	acetylcholine M1, M2 , M3 receptors and M4, which is bel
act as inhibitors of the neurotransmitter	acetylcholine, may provide some relief.
testinal tract by blocking the activity of	acetylcholine on cholinergic (or muscarinic) receptors o
by antagonism of the excitatory effects of	acetylcholine on the cortex, reduction of locomotor acti
inity for adrenaline, dopamine, muscarinic	acetylcholine, or serotonin receptors, or any of the mon
turnover number was 3 x 107 (molecules of	acetylcholine) per minute per molecule of enzyme.
erase hence increases the concentration of	acetylcholine present).
le to a neuron will decrease the amount of	acetylcholine produced.
th the nootropic effect from the increased	acetylcholine production cancelled out by the opposite e
acetyltransferase, resulting in increased	acetylcholine production.
It is a nicotinic	acetylcholine receptor antagonist.
Aceclidine acts as a muscarinic	acetylcholine receptor agonist.
also mildly anticholinergic, or muscarinic	acetylcholine receptor antagonistic.
It also acts as a nicotinic	acetylcholine receptor antagonist.
CHRNA7, coding the neuronal nicotinic	acetylcholine receptor alpha7 subunit.
ts mode of action is through the nicotinic	acetylcholine receptor where it acts as an analogue of a
An	acetylcholine receptor (abbreviated AChR) is an integral
The	acetylcholine receptor changes conformation upon acetylc
by this gene is a subunit of the nicotinic	acetylcholine receptor (nAchR).
ylphenylpiperazinium (DMPP) is a nicotinic	acetylcholine receptor agonist which is selective for th
nt, noncompetitive α3β4 neuronal nicotinic	acetylcholine receptor antagonist.
	Acetylcholine receptor subunit delta is a protein that i
racetam family, and an antidepressant (M1	acetylcholine receptor agonist).
	Acetylcholine receptor subunit beta is a protein that in
ism of action is to bind to the muscarinic	acetylcholine receptor M1.
Neuronal	acetylcholine receptor subunit alpha-7 is a protein that
Cytisine is a nicotinic	acetylcholine receptor agonist, and as a pharmaceutical
Structure of the nicotinic	acetylcholine receptor (nAchR: PDB 2BG9) which is very s
Neuronal	acetylcholine receptor subunit alpha-2 is a protein that
Neuronal	acetylcholine receptor subunit alpha-3 is a protein that
Neuronal	acetylcholine receptor subunit alpha-9 is a protein that
d-gated ionic channel family and nicotinic	acetylcholine receptor gene superfamily.
Neuronal	acetylcholine receptor subunit beta-3 is a protein that
Neuronal	acetylcholine receptor subunit alpha-10 also known as ch
lly moderately to highly potent muscarinic	acetylcholine receptor (anticholinergic) antagonists as
onist, with specificity for the Muscarinic	acetylcholine receptor M2, made it a promising starting
uscle, where it may regulate agrin-induced	acetylcholine receptor clustering at the neuromuscular j
inds irreversibly and competitively to the	acetylcholine receptor found at the neuromuscular juncti
a selective antagonist of the α7 nicotinic	acetylcholine receptor in the brain, and as such has app
The	acetylcholine receptor of muscle has 5 subunits of 4 dif
its antihistamine effects, and muscarinic	acetylcholine receptor antagonism is responsible for its
Since agrin fragments induce	acetylcholine receptor aggregation as well as phosphoryl
The muscle	acetylcholine receptor is composed of five subunits: two
-246,708; Lumeron, Memcor) is a muscarinic	acetylcholine receptor agonist with reasonable selectivi
amine acts in vitro as a strong muscarinic	acetylcholine receptor antagonist (anticholinergic) and
e (WAL-2014) is a non-selective muscarinic	acetylcholine receptor agonist which acts as a full agon
d that development of new neural nicotinic	acetylcholine receptor agonists will be likely to lead t
as selective potentiator of α4β2 nicotinic	acetylcholine receptor responses by using two-electrode
allosteric modulator of neuronal nicotinic	acetylcholine receptor with sub-type specificity for het
d-gated ion channels such as the nicotinic	acetylcholine receptor and GABAA receptor are composed o
ses the actions of the vagus nerve, blocks	acetylcholine receptor sites, and decreases bronchial se
n, adrenenaline, histamine, and muscarinic	acetylcholine receptor antagonist, though with weaker an
cking drugs in a long lineage of nicotinic	acetylcholine receptor anatagonists synthesized by Mary
The	Acetylcholine receptor Pathways
enacin works by blocking the M3 muscarinic	acetylcholine receptor, which is primarily responsible f
nicotinic receptor is a type of nicotinic	acetylcholine receptor, consisting of the subunit combin
nicotinic receptor is a type of nicotinic	acetylcholine receptor, consisting of the subunit combin
the α3β4 receptor, is a type of nicotinic	acetylcholine receptor, consisting of α3 and β4 subunits
This gene encodes the beta subunit of the	acetylcholine receptor.
osely related by homology to the nicotinic	acetylcholine receptor.
, nicotine acts primarily on the nicotinic	acetylcholine receptor.
for the α2β2 subtype of neuronal nicotinic	acetylcholine receptor.
ect causes "fast channel" behaviour of the	acetylcholine receptor.
nicotinic	acetylcholine receptors (nAChR, also known as "ionotropi
muscarinic	acetylcholine receptors (mAChR, also known as "metabotro
cts of GHB are mediated through muscarinic	acetylcholine receptors and can be prevented by prior ad
α-Bungarotoxin blocks nicotinic	acetylcholine receptors and has been used to isolate and
d on its involvement in the aggregation of	acetylcholine receptors during synaptogenesis.
Muscarinic	acetylcholine receptors belong to a class of metabotropi
function of its competitive antagonism to	acetylcholine receptors of the nicotinic type.
inhibition of both α2β2 and α4β2 Nicotinic	Acetylcholine Receptors by β-Amyloid (1-42) Peptide .
nergic medication that inhibits muscarinic	acetylcholine receptors and thus the parasympathetic ner
ich acts as an agonist at neural nicotinic	acetylcholine receptors with high selectivity for the α4
The nicotinic	acetylcholine receptors (nAChRs) are members of a superf
hermore selective block of α9α10 nicotinic	acetylcholine receptors by the conotoxin RgIA has been s
ceptors with similar potency and nicotinic	acetylcholine receptors to a much lesser extent.
hycanthone binds to	acetylcholine receptors in the worm and result inincreas
arch to map the distribution of muscarinic	acetylcholine receptors in the brain.
n identified as an antagonist of nicotinic	acetylcholine receptors (nAChrs) in the muscle and brain
as a mixed agonist-antagonist at nicotinic	acetylcholine receptors and an antagonist at μ-opioid re
the H1 receptor (24,000 nM) or muscarinic	acetylcholine receptors (11,000 nM), and accordingly lac
nM) > H1 receptor (3,100 nM) > muscarinic	acetylcholine receptors (>35,000).
Selective block of α9α10 nicotinic	acetylcholine receptors by the conotoxin RgIA has been s
ts on serotonin, histamine, and muscarinic	acetylcholine receptors have not been assayed, but uniqu
does not have any effect on the muscarinic	acetylcholine receptors (mAChR) located on target organs
ich acts as an agonist at neural nicotinic	acetylcholine receptors selective for the α4β2 subtype,
and 18-methoxycoronaridine block nicotinic	acetylcholine receptors in the brain and can be used for
that also includes the neuronal nicotinic	acetylcholine receptors (nAChRs), and the inhibitory neu
ich acts as an agonist at neural nicotinic	acetylcholine receptors and has been researched for use
arch to map the distribution of muscarinic	acetylcholine receptors in the brain, however this drug
Main article: Muscarinic	acetylcholine receptors
ct of the poisoning by blocking muscarinic	acetylcholine receptors, which would otherwise be overst
vates, and desensitizes neuronal nicotinic	acetylcholine receptors, though at much lower potency in
when the body produces antibodies against	acetylcholine receptors, and thus inhibits signal transm
s as a partial agonist at neural nicotinic	acetylcholine receptors, binding to both the α3β4 and th
ohol because alcohol potentiates nicotinic	acetylcholine receptors, leading to re-sensitization and
ist for the α7 subtype of neural nicotinic	acetylcholine receptors, with a high level of brain pene
as a partial agonist at neuronal nicotinic	acetylcholine receptors, binding to both the α3β4 and th
receptors, opioid receptors, and nicotinic	acetylcholine receptors, but not benzodiazepinergic or m
n including antagonist action at nicotinic	acetylcholine receptors, and it has also been shown to r
receptors, histamine receptors, muscarinic	acetylcholine receptors, and serotonin receptors.
potent and selective agonist for nicotinic	acetylcholine receptors, with twice the potency of nicot
e partial agonist at α4β2 neural nicotinic	acetylcholine receptors, acting as an agonist at (α4)2(β
s a mixed agonist-antagonist at muscarinic	acetylcholine receptors, being a potent and selective ag
rgic that inhibits the action at nicotinic	acetylcholine receptors.
y through dopamine modulation by nicotinic	acetylcholine receptors.
t's nervous system by inhibiting nicotinic	acetylcholine receptors.
er, α2-adrenergic receptor, and muscarinic	acetylcholine receptors.
s as a partial agonist at neural nicotinic	acetylcholine receptors.
for the D2, 5-HT2, H1, H2, and muscarinic	acetylcholine receptors.
s as a partial agonist at neural nicotinic	acetylcholine receptors.
ist for the α7 subtype of neural nicotinic	acetylcholine receptors.
ist for the α7 subtype of neural nicotinic	acetylcholine receptors.
ist for the α7 subtype of neural nicotinic	acetylcholine receptors.
ist for the α7 subtype of neural nicotinic	acetylcholine receptors.
lated by nicotinic ligand-gated ionotropic	acetylcholine receptors.
eceptors, without affecting the muscarinic	acetylcholine receptors.
amine receptors, as well as the muscarinic	acetylcholine receptors.
as an antagonist towards various nicotinic	acetylcholine receptors.
ny of the TCAs analyzed for the muscarinic	acetylcholine receptors.
is of a radiotracer for studying nicotinic	acetylcholine receptors: (+/-)-Exo-2-(2-[F-18]fluoro-5-p
It enhances	acetylcholine release through indirect activation of the
ins depolarized and unresponsive to normal	acetylcholine release.
elease, as well as modulating dopamine and	acetylcholine release.
Similarly,	acetylcholine released from parasympathetic neurons may
It hydrolyzes	acetylcholine to choline and an acetate group.
he NMJ nAChRs by preventing the binding of	acetylcholine to its receptor.
Binding of	acetylcholine to the N termini of each of the two alpha
ry neurotransmitters such as glutamate and	acetylcholine via stimulation of H1 receptors in the cer
stimulation, these nerve branches release	acetylcholine, which, in turn causes release of nitric o

共起表現

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「ACETYLCHOLINE」の共起表現一覧(1語右で並び替え)