「HIV-1」の共起表現一覧(1語右で並び替え)
該当件数 : 45件
CX3CR1 also is a coreceptor for | HIV-1, and some variations in this gene lead to incr |
These include human immunodeficiency viruses ( | HIV-1 and HIV-2) and simian immunodeficiency virus ( |
cells of the human genital epithelium binds to | HIV-1 and subsequently internalizes it into Birbeck |
resses structural analysis of the AIDS viruses | HIV-1 and SIV, and the antibodies that neutralize th |
receptor for the human immunodeficiency virus, | HIV-1) and CD8 on T-cells interact with protein-tyro |
t (scr2), or as a potential human repressor of | HIV-1 and ILR-2 alpha promoter transcription (YC1). |
It can be found in | HIV-1 but not in HIV-2. |
MCP-2 is a potent inhibitor of | HIV-1 by virtue of its high-affinity binding to the |
HIV-1 carries out reverse transcription, a process t | |
This is a solution RNA structure model of the | HIV-1 dimerization initiation site in the kissing-lo |
The | HIV-1 DIS structure consists of a conserved hairpin |
ncy virus type 1 dimerization initiation site ( | HIV-1 DIS) is an RNA structure that is shown to prom |
f Michigan determined that BanLec bound to the | HIV-1 envelope protein gp120, which is high in sugar |
change in a single gene (Trim-5alpha) prevents | HIV-1 from replicating in monkeys. |
s found to be an important cellular factor for | HIV-1 gene expression and viral replication. |
that is known to direct specific packaging of | HIV-1 genomic RNA. |
des, carbohydrates and viral proteins, such as | HIV-1 gp120 and gp41. |
intercellular adhesion molecule 3 (ICAM3) and | HIV-1 gp120, and enhances HIV-1 infection of T cells |
"Hit | HIV-1 hard, but only when necessary", Mike Harringto |
s a host defence to retroviral infection which | HIV-1 has overcome by the acquisition of Vif. |
cDNA is also produced by retroviruses (such as | HIV-1, HIV-2, Simian Immunodeficiency Virus, etc.) w |
A lectin (BanLec) from bananas inhibits | HIV-1 in vitro |
her antiretroviral agents for the treatment of | HIV-1 infection in adults. |
atment of human immunodeficiency virus type 1 ( | HIV-1) infection in antiretroviral treatment-experie |
d response to HAART during initial therapy for | HIV-1 infection" (2007) Journal of Psychosomatic Res |
Enfuvirtide is indicated for the treatment of | HIV-1 infection, in combination therapy with other a |
to free radicals, and inhibit the function of | HIV-1 integrase. |
5HN brings integrated | HIV-1 out of latency but does not activate all T cel |
HIV-1 progression can be delayed by using Aciclovir, | |
The structure of | HIV-1 protease complexed with inhibitor BEA369 based |
The structure of | HIV-1 protease (green and cyan) complexed with a pol |
It was found to inhibit | HIV-1 replication in vitro and during Phase II clini |
the nuclear export of many proteins including | HIV-1 Rev, MAPK/ERK, and NF-κB/IκB, and it stabilize |
Proposed | HIV-1 RNase H mechanism |
Within human immunodeficiency virus type 1 ( | HIV-1), RNase H exists as a domain in the heterodime |
The | HIV-1 SD structure consists of a highly conserved 19 |
unodeficiency virus type 1 major splice donor ( | HIV-1 SD) is an RNA structure that is shown to modul |
nfection by targeting and crippling the sticky | HIV-1 spike proteins. |
bit the replication of the clinically isolated | HIV-1 strain as well as the cultured strain in vitro |
mple with the histone acetyltransferase TIP60 ( | HIV-1 Tat interacting protein 60 kDa), which acetyla |
eptors are required for internalization of the | HIV-1 tat protein. |
nevitable consequence of prolonged exposure of | HIV-1 to antiretroviral therapy. |
2 (or SDF-1) and, as with CCR5, is utilized by | HIV-1 to gain entry into target cells. |
a 2 year trial, 284 people progressed with the | HIV-1, versus 324 who had not been treated with Acic |
to be recruited by the Gag polyprotein during | HIV-1 virus infection, and its incorporation into ne |
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