「MYELOID」の共起表現(1語右で並び替え) - Weblio英語共起表現検索


小窓モード

プレミアム

ログイン
設定

設定

「MYELOID」の共起表現一覧(1語右で並び替え)

該当件数 : 52



chest weakness, anaemia, some suppression of myeloid activity and abdominal pains.
Myeloid and erythroid nuclear termination stage-speci
gene is expressed in B-cell but not T-cell or myeloid cell lines.
greatly induced under conditions suitable for myeloid cell differentiation.
e and rat suggest that this gene may regulate myeloid cell activity and delivers an inhibitory sign
, endothelial cells, platelets, monocytes and myeloid cell lines, blood T-lymphocytes and B-lymphoc
h affinity counter-receptor for P-selectin on myeloid cells and stimulated T lymphocytes.
is reported to promote survival of progenitor myeloid cells through the insulin receptor substrate-
rotein is responsible for preventing immature myeloid cells from differentiating into more mature c
kin grafts upon ligation of CD200 receptor on myeloid cells
inhibitory CD32B is expressed on B cells and myeloid dendritic cells.
ene suggest that this interaction may control myeloid function in a tissue-specific manner.
Spi-1 regulates myeloid gene expression during haemopoietic developme
in mammals, CALM, (Clathrin-assembly lymphoid myeloid leukaemia protein) is named after its associa
are among the most common mutations in acute myeloid leukaemia due to internal tandem duplication
bine is mainly used in the treatment of acute myeloid leukaemia, acute lymphocytic leukaemia (ALL)
ncipal use is in acute leukaemias and chronic myeloid leukaemia.
ced that Todd had been diagnosed with chronic myeloid leukaemia.
under development for the treatment of acute myeloid leukaemia.
-inducing agent in malignant glioma and acute myeloid leukaemia.
ity to induce the terminal differentiation of myeloid leukaemic cells.
to be administered in the diagnosis of acute myeloid leukemia to demonstrate that the leukemic cel
A potential link to leukemia including acute myeloid leukemia has also been described.
hibitor undergoing clinical studies for acute myeloid leukemia (AML) and myelodysplastic syndrome (
being investigated for the treatment of acute myeloid leukemia (AML) and myelodysplastic syndrome (
s of age and older with newly diagnosed acute myeloid leukemia (AML).
CR-ABL inhibitor for the treatment of chronic myeloid leukemia (CML).
When diagnosed with acute myeloid leukemia and informed that traditional treatm
by Seattle Genetics as a treatment for acute myeloid leukemia (AML), a condition which results in
exemplified by their treatment-related acute myeloid leukemia (AML), which is uncommon for antimet
mmer for The Brakes, was diagnosed with acute myeloid leukemia in August, 2007 and he died on May 4
cell surface antigen expressed by CD34+ acute myeloid leukemia cell lines, T-cell lines, activated
may increase risk for myelofibrosis and acute myeloid leukemia in the long term.
or of Bcr-Abl kinase induces death of chronic myeloid leukemia (CML) cells.This work led to the dev
s laevis proteins Xr1 and Xr11; human induced myeloid leukemia cell differentiation protein MCL1 an
it was reported that Burr was diagnosed with myeloid leukemia, which may require a bone marrow tra
ancer, mostly metastatic breast cancer, acute myeloid leukemia, and non-Hodgkin's lymphoma.
In acute myeloid leukemia, especially in the M2 subtype, the t
is a common site of retroviral integration in myeloid leukemia, and may function as a leukemia dise
ing this gene have been associated with acute myeloid leukemia.
has an inherent risk of transforming to acute myeloid leukemia.
his receptor is restricted to the surfaces of myeloid lineage cells and the receptor-substrate inte
mbrane glycoprotein present on the surface of myeloid lineage cells such as neutrophils, monocytes,
against JAK2 for relapsed lymphoma, advanced myeloid malignancies, myelofibrosis and CIMF
l abnormality associated with therapy-related myeloid malignancies.
cythemia vera, essential thrombocythemia, and myeloid metaplasia with myelofibrosis.
He died in 1967 of myeloid metaplasia, a rare blood disease.
xpression also occur in certain cell lines of myeloid origin (e.g. the immature myeloid cell lines
They differentiate from myeloid precursor cells in response to the cytokines
nduction of growth arrest and/or apoptosis of myeloid precursor cells
1), although the underlying genetic defect in myeloid precursor cells is not entirely elucidated, m
T cells, activated B cells, some thymocytes, myeloid precursors, and oligodendrocytes that associa
                                                                                                   


こんにちは ゲスト さん

ログイン

Weblio会員(無料)になると

会員登録のメリット検索履歴を保存できる!

会員登録のメリット語彙力診断の実施回数増加!

無料会員に登録する
英→日 日→英
こんにちは ゲスト さん

ログイン

Weblio会員(無料)になると

会員登録のメリット検索履歴を保存できる!

会員登録のメリット語彙力診断の実施回数増加!

無料会員に登録する

©2024 GRAS Group, Inc.RSS