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「NMDA」の共起表現一覧(1語左で並び替え)

該当件数 : 93



Huperzine A is also a NMDA receptor antagonist which protects the brain ag
as a neuronal signaling molecule by activating NMDA receptors in the brain.
sions") occurred in brains of rats administered NMDA antagonists, including PCP, MK-801 (dizocilpine
decahydroquinoline (8A-PDHQ) is a high affinity NMDA antagonist.
It also acts as an NMDA receptor antagonist.
estinel (GV-150,526) is a drug which acts as an NMDA antagonist, binding selectively to the glycine
esthetic and pharmacologically classified as an NMDA receptor antagonist.
esonprodil (CI-1041) is a drug which acts as an NMDA antagonist, selective for the NR2B subunit.
iprodil (SL-82.0715) is a drug which acts as an NMDA antagonist, binding to the polyamine modulatory
elucemine (NPS-1506) is a drug which acts as an NMDA antagonist and a serotonin reuptake inhibitor,
s a drug related to memantine, which acts as an NMDA antagonist and has neuroprotective effects.
It acts as an NMDA receptor antagonist but has low potency and act
is a drug developed by Pfizer which acts as an NMDA antagonist, selective for the NR2B subunit.
As an NMDA receptor antagonist, in vivo, it is approximate
nervous system and has its mode of action as an NMDA receptor antagonist.
ome apparent 2 hours after administration of an NMDA antagonist.
tive than 18-MC with very little activity as an NMDA antagonist and only slight affinity for the del
eceptor agonist, κ-opioid receptor agonist, and NMDA receptor antagonist.
ated ion channels and include AMPA, Kainate and NMDA.
ls and has a low affinity for AMPA, kainate and NMDA receptors.
ol's acute effects on GABAergic enhancement and NMDA suppression, leading to CNS depression leading
A and 5-HT2C), 5-HT6, 5-HT7, α1-adrenergic, and NMDA receptors, and as agonists at the sigma recepto
ors it has been demonstrated to function on are NMDA and the D1 dopamine receptor.
oxic changes corresponded with their potency as NMDA antagonists: i.e.
ther ionotropic receptors for glutamate such as NMDA and AMPA.
neurotrophic factor (BDNF), glutamate acting at NMDA receptors, dopamine through activation of the D
zoline I2 receptor antagonists reversibly block NMDA receptor-mediated Ca2+ influx and thus may inhi
ing within in vitro preparations, and can block NMDA receptor action at a reasonably small concentra
eptor and imidazoline binding sites, and blocks NMDA receptors and other cation ligand-gated channel
trorotary isomer, dextromethorphan, is a common NMDA receptor antagonist and pro-drug to dextrorphan
GS-19755) is a drug which acts as a competitive NMDA antagonist, directly competing with glutamate f
and that the cortical NMDA receptor properties and membrane fluidity are a
rent ability to stabilize disrupted or disabled NMDA receptor pathways in the brain, which are share
I added dissociatives ( NMDA antagonists) as psychedelics.
ensory nerve terminals in the skin also express NMDA and non-NMDA receptors.
ying ovarian or mediastinal teratoma expressing NMDA receptors.
ty of several ion channels including the GABAA, NMDA, and sigma receptors.
Glutamate [ NMDA] receptor subunit 3A is a protein that in human
Glutamate [ NMDA] receptor subunit epsilon-1 is a protein that i
Glutamate [ NMDA] receptor subunit epsilon-4 is a protein that i
Glutamate [ NMDA] receptor subunit 3B is a protein that in human
") and ketamine, both of which block glutamate ( NMDA) receptors, are known to cause psychosis closel
Dextrorphan - A dissociative hallucinogen ( NMDA receptor antagonist) and cough suppressant.
.3x meperidine in potency) although it also has NMDA antagonist properties like its close relative k
ulators of a variety of ion channels, including NMDA receptors and AMPA receptors.
apse to an active one, since this conversion is NMDA receptor-dependent.
Its NMDA actions, similar to those of the phenylheptylam
Lacosamide does not affect AMPA, kainate, NMDA, GABAA, GABAB or a variety of dopaminergic, ser
It has also been shown to act as a low-affinity NMDA receptor antagonist.
-physiological partial agonist of the mammalian NMDA receptor.
In animal models, NMDA antagonists increase glutamate release in the p
development of synaptic elements by modulating NMDA receptor activity.
Most NMDA receptor antagonists are metabolized in the liv
Frequent administration of most NMDA receptor antagonists can lead to tolerance, whe
N-methyl-D-aspartate ( NMDA) receptors are a class of ionotropic glutamate
ith different subunits of N-methyl-D-aspartate ( NMDA) receptor.
encodes a subunit of the N-methyl-D-aspartate ( NMDA) receptors, which belong to the superfamily of
It is a non-competitive NMDA antagonist and calcium channel blocker
e AMPA receptor antagonist, and non-competitive NMDA receptor antagonist.
ts and instead acts as a potent non-competitive NMDA antagonist.
-1102) is a drug which acts as a noncompetitive NMDA antagonist.
able AMPA receptor and kainate receptor but not NMDA receptor.
d her colleagues demonstrated the importance of NMDA receptors in development plasticity.
The induction of NMDA receptor-dependent long-term potentiation (LTP)
A class of NMDA receptor antagonists have been denoted dissocia
holine antagonists, with little or no effect on NMDA receptors.
Glutamate opens NMDA receptors on other neurons which allows calcium
MK-801, along with other NMDA antagonists, induce the formation of brain lesi
Other NMDA receptor modulators are being studied and this
otel (EAA-090) is a drug which acts as a potent NMDA antagonist.
de of GlyT1 has been hypothesized to potentiate NMDA receptor function in vivo and to represent a ra
(Tet-Gly, LY-285,265) is a potent and selective NMDA receptor agonist, stimulating the NMDA receptor
2R)-amino-5-phosphonopentanoate) is a selective NMDA receptor antagonist that competitively inhibits
AP-7 is a selective NMDA receptor (NMDAR) antagonist that competitively
CKA (5,7-dichlorokynurenic acid) is a selective NMDA receptor (NMDAR) antagonist acting at the glyci
ffects on Aplysia long-term potentiation, since NMDA receptors are required for both.
r treatment focus because glutamate blocks some NMDA receptors which, on their own, induce schizophr
l experiments, Palazzo et al. demonstrated that NMDA, an ionotropic glutamate receptor, and the meta
PEAQX is a competitive antagonist at the NMDA receptor.
Chemicals that deactivate the NMDA receptor are called antagonists.
should be drafted for the establishment of the NMDA.
Examples include haemoglobin (pictured), the NMDA receptor, some aquaporins, some AMPA receptors,
dies of glycine (and related co-agonists at the NMDA receptor) added to conventional anti-psychotics
an receptor site is the PCP binding site in the NMDA receptor, a type of ligand-gated ion channel.
antibodies to the delta2 or NR2 subunits of the NMDA receptor.
kappa and delta opioid, sigma receptors, or the NMDA system are not well understood.
red in combination with other drugs such as the NMDA antagonist tiletamine or the alpha-2 adrenergic
After repeated doses of the NMDA antagonists MK-801 and PCP, the vacuolation rea
stulated that this is a homeostatic response to NMDA receptor blockade, which in turn increases psyc
as increased glucose metabolism in response to NMDA antagonist exposure.
t a hypofunction of glutamatergic signaling via NMDA receptors.
a number of receptor systems, being both a weak NMDA antagonist and a 5HT1A agonist.
antidepressant effect when co-administered with NMDA antagonists.
                                                                                                    


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