「THROMBIN」の共起表現一覧(1語右で並び替え)
該当件数 : 36件
| ctive antithrombins no longer able to inhibit | thrombin activity. |
| he proteins that cleave prothrombin to active | thrombin after injury. |
| eases like chymotrypsin, kallikrein, plasmin, | thrombin, and trypsin. |
| Expression of TF after | thrombin and S1P stimulation activates the coagulation |
| ivate the "final common pathway" of factor X, | thrombin and fibrin. |
| red for the efficient production of activated | thrombin and also for adequate hemostasis. |
| ctor (TF) is induced by the interplay between | thrombin and sphingosine 1-phosphate (S1P). |
| Other than antibodies to | thrombin, antibodies to vascular heparin sulfate appea |
| Calcium and the enzyme | thrombin are added, forming a gel that is high in natu |
| Antibodies (IgG) against | thrombin can strongly inhibit its activity. |
| e activated by molecules such as histamine or | thrombin during inflammation, P-selectin moves from an |
| Protein C Anticoagulant Pathway: | Thrombin escaping from a site of vascular injury binds |
| with the prothrombinase complex, the rate of | thrombin formation is severely decreased under such ci |
| Cofactor II deficiency can lead to increased | thrombin generation and a hypercoagulable state. |
| or VIIIa, which allows for longer duration of | thrombin generation and may lead to a hypercoagulable |
| n stage 2, the amplification stage, if enough | thrombin has been generated, then activation of platel |
| ombin antibodies can react with both types of | thrombin in the antithrombin-thrombin complex. |
| e proteinase inhibitor which rapidly inhibits | thrombin in the presence of dermatan sulfate or hepari |
| Indeed, | thrombin inhibition after injury to the aorta has been |
| vity is inhibited by hirudin and other direct | thrombin inhibitors, but not by heparin. |
| owever, that despite autoimmune anti-thrombin | thrombin inhibitory activity, these antibodies correla |
| Given time, | thrombin is able to cleave the reactive bond within an |
| The activation of | thrombin is a critical reaction in the coagulation cas |
| Thrombin is one trigger which can stimulate endothelia | |
| As a result, | thrombin loses its procoagulant properties and instead |
| but an analog is now in clinical trials as a | thrombin receptor antagonist. |
| ), previously known as Coagulation factor II ( | thrombin) receptor-like 2, is a protein encoded by the |
| Coagulation factor II ( | thrombin) receptor-like 2 (F2RL2) is a member of the l |
| receptor that is most closely related to the | thrombin receptor. |
| ndsidered different from antibodies that bind | thrombin, serum phospholipids and are called anti-apol |
| ntibodies are autoantibodies directed against | thrombin that may constitute a fraction of lupus antic |
| f the tissue factor pathway is to generate a " | thrombin burst," a process by which thrombin, the most |
| To produce | thrombin, the prothrombinase complex cleaves two pepti |
| rtial thromboplastin time), prothrombin time, | thrombin time and fibrinogen level. |
| s, like fibrin degradation products, D-dimer, | thrombin time, platelet aggregation, or |
| ime' as a heparin-insensitive parallel to the | thrombin time, to investigate dysfibrinogenemia, and t |
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