「cancer」の共起表現一覧(1語右で並び替え)5ページ目
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| pporting charities including Refuge and Breast | Cancer Care. |
| Dana Farber/Partners | Cancer Care: Collaborative program for cancer care, i |
| c violence charity Women's Aid and of Maggie's | Cancer Caring Centre, set up in memory of a breast ca |
| bear the brunt of an estimated 27 million new | cancer cases and 17 million cancer deaths. |
| About extreme terminal | cancer cases, Contreras said: "The palliative action |
| ollution, and genetics account for 10% of lung | cancer cases...so maybe not. |
| led to the development of drugs to treat Aids, | cancer, cataracts and diabetes. |
| Member, House | Cancer Caucus |
| He also announced the Pediatric | Cancer Caucus, which he will co-chair. |
| transfer of viruses, in the manner of cervical | cancer caused by HPV. |
| In 1983, Barger was diagnosed with throat | cancer, caused by years of heavy smoking. |
| one of the pioneers of radioactivity, died of | cancer caused by radiation exposure during her experi |
| The | cancer caused intense pain, which she reportedly bore |
| king (particularly burning) of meat forms some | cancer causing heterocyclic amines. |
| ms a shell-shocked veteran that he is dying of | cancer, causing the distraught man to commit suicide. |
| lling but also mediates increased human breast | cancer cell spread. |
| Cancer Cell Int. | |
| the recent commercialization of isogenic human | cancer cell disease models for the pharmaceutical ind |
| s shown to stimulate estrogen-dependent breast | cancer cell growth in vivo. |
| "Sweet Oddysee of a | Cancer Cell t' th' Center of Yer Heart" - 7:41 (5:27) |
| It has shown anti-cancer activity in human | cancer cell lines. |
| bacterial antibiotic migrastatin, which stops | cancer cell metastasis in some animal models. |
| production of hPR-B in an Ishikawa endometrial | cancer cell line. |
| 98 shows that anandamide inhibits human breast | cancer cell proliferation. |
| trials, it demonstrated cytotoxic activity in | cancer cell lines that had either intrinsic or acquir |
| Cell growth and division ( | cancer, cell cycle, cytokinesis) |
| hat their cytotoxicity and selectivity towards | cancer cell is higher than that of cisplatin. |
| eported that dFBr was cytotoxic on human colon | cancer cell line HCT-116. |
| This gene is highly expressed in two human | cancer cell lines, indicating that it may play a role |
| sine kinase receptor, is overexpressed by many | cancer cell types. |
| luding an NIH merit award from the division of | cancer cell biology. |
| i, and inhibits the proliferation of different | cancer cell lines including a multidrug-resistant KB |
| acent endothelial cells to fail and allows the | cancer cell to slip through. |
| - cannabidiol, has also been shown to inhibit | cancer cell growth, with low potency in non-cancer ce |
| When LNCaP cells (a prostate | cancer cell line) were treated with MDV3100, the expr |
| In the case of breast | cancer cell lines, the ability to form invadopodia is |
| BIBW 2992 inhibits survival of | cancer cell lines and induces tumour regression in xe |
| these systems, HA produced by HAS protects the | cancer cell from physical damage. |
| d to latch onto the surface glycoproteins of a | cancer cell, allowing it to enter and destroy the cel |
| copy: Single YFP molecule detection in a human | cancer cell. |
| so able to kill human leukemic cells and colon | cancer cells by apoptosis. |
| Cancer cells frequently have aneuploidy of chromosome | |
| f killing GPNMB expressing melanoma and breast | cancer cells in vitro and inducing partial or complet |
| ntified a way to exploit genetic weaknesses in | cancer cells including mutated BRCA 1 or BRCA2, leadi |
| N-Cadherin is commonly found in | cancer cells and provides a mechanism for transendoth |
| of PCGEM1 were found to be higher in prostate | cancer cells in African-American men than in Caucasia |
| The enforced expression causes the growth of | cancer cells to slow down. |
| Transfection of breast | cancer cells with ferroportin significantly reduces t |
| stically act with sirolimus to kill pancreatic | cancer cells in the laboratory in a Mayo Clinic study |
| idar inhibits this P-glycoprotein, causing the | cancer cells to lose their medicine tolerance, and ma |
| onsible for the invasive properties of hypoxic | cancer cells through focal adhesion kinase activity a |
| Many types of | cancer cells carry EpCAM (epithelial cell adhesion mo |
| es cytochrome c release in the mitochondria of | cancer cells leading to cell death, but does not harm |
| aller extent, testosterone, stimulate prostate | cancer cells to grow. |
| CBD reduces growth of aggressive human breast | cancer cells in vitro and reduces their invasiveness. |
| It slows or stops the growth of | cancer cells in the body. |
| may have an anti-proliferative effect on colon | cancer cells and an anti-diabetic effect by means of |
| an antibody-drug conjugate (ADC) that targets | cancer cells expressing transmembrane glycoprotein NM |
| CD47 is used by bladder | cancer cells to hide from normal scavenging by macrop |
| found that the overexpression of H19 in breast | cancer cells promotes proliferation. |
| t cancers as protective factors for the breast | cancer cells makes them important targets for structu |
| oproteinases, a function important to invading | cancer cells within bone. |
| In human | cancer cells DNMT1 is responsible for both de novo an |
| Prototypes that separate | cancer cells from healthy cells have been made. |
| cin is able to trigger apoptosis in human lung | cancer cells as well. |
| ction is that the compound binds to the DNA of | cancer cells rendering them incapable of dividing. |
| ed by microwave irradiation to damage and kill | cancer cells or to make cancer cells more sensitive t |
| egulation of estrogen receptor negative breast | cancer cells transfected with cDNA's for estrogen rec |
| Removing | cancer cells from the abdominal cavity reduces the tu |
| Cancer cells generally divide more often than healthy | |
| Splice variants of CD44 on colon | cancer cells display sialofucosylated HCELL glycoform |
| c gene variations, or molecular signatures, in | cancer cells and use that knowledge to accelerate the |
| type in estrogen receptor (ER)-positive breast | cancer cells with deficient intercellular adhesion. |
| e high levels of SP2 protein that are found in | cancer cells would be neutralized. |
| It is highly potent agent, capable of damage | cancer cells in very low concentrations, and retains |
| hyllotoxin derivatives and slows the growth of | cancer cells in the body. |
| ltimately leads to apoptosis in WT human colon | cancer cells but not in human colon cancer cells lack |
| It is found on the surface of many types of | cancer cells and shed into the blood stream. |
| ignaling and are frequently seen in metastatic | cancer cells that are invading surrounding tissues. |
| Effect on Multi Drug Resistant | Cancer Cells |
| It acts to freeze | cancer cells in phase G1 of the cell cycle, preventin |
| It works by damaging DNA, primarily in | cancer cells that preferentially take up the uracil d |
| to mesothelin (a protein that is made by some | cancer cells) and stops the cells from dividing. |
| s (an immortal cell line derived from cervical | cancer cells), but was later found to be ubiquitously |
| fect all rapidly dividing cell types including | cancer cells, but also those of intestinal epithelium |
| ant role in the immune system's recognition of | cancer cells, so indiscriminate blocking of IgE / rec |
| in fusion transcripts are commonly produced by | cancer cells, and detection of fusion transcripts is |
| ic acid was found to show cytotoxicity against | cancer cells, without harming healthy cells. |
| p53-deficient human gastric | cancer cells, restoration of functional miR-34 inhibi |
| The antibody binds to GPNMB on the | cancer cells, the ADC is internalised, the linkage is |
| ve pro-adhesive and chemotactic activities for | cancer cells, suggesting that this molecule may have |
| similarities in the gene pathways activated in | cancer cells, and those activated in cells involved i |
| ts participate in the selective destruction of | cancer cells, or inhibit the growth and proliferation |
| increased proliferation and migration of colon | cancer cells, which might encourage cancer cells to m |
| n coded with the MDR1 gene manifests itself in | cancer cells, it discharges much of the antineoplasti |
| Importantly, vitamin D treatment of colon | cancer cells, Caco-2, also increases expression of TR |
| genetically-enhanced virus based on Ebola and | cancer cells, which they plan to use to wipe out much |
| serve as adhesion molecule for lung metastatic | cancer cells, mediating vascular arrest and colonizat |
| he inhibition of the proliferation of prostate | cancer cells, have also been investigated. |
| In | cancer cells, EP300 mutations prevent the gene from p |
| proven ability to inhibit energy metabolism in | cancer cells, and to enhance the activity of anticanc |
| hesis and DNA repair in both healthy cells and | cancer cells. |
| Expression of MEG3 is lost in | cancer cells. |
| lations ) in HIV infected monocytes and breast | cancer cells. |
| it shows anticancer activity against prostate | cancer cells. |
| shows anti-cancer activity against human colon | cancer cells. |
| ng a buildup of reactive oxygen species within | cancer cells. |
| triggers apoptosis (programmed cell death) in | cancer cells. |
| preventing the replication of rapidly-growing | cancer cells. |
| s) caused apoptosis (programmed cell death) in | cancer cells. |
| or using the body's own immune system to fight | cancer cells. |
| is in normal cells, but suppress glycolysis in | cancer cells. |
| posedly studying gene therapy and immunity for | cancer cells. |
| and bind to substances in the body, including | cancer cells. |
| ving enzyme with toxicity that is specific for | cancer cells. |
| h mammalian type, eucariotic topoisomerases in | cancer cells. |
| a growth-inhibiting state called senescence in | cancer cells. |
| cts of dihydroxycholecalciferol in human colon | cancer cells. |
| d with the over-expression of oncogenes within | cancer cells. |
| agent with growth-inhibitory effects in human | cancer cells. |
| n (CEA), a protein present on certain types of | cancer cells. |
| nces cell motility, invasion and metastasis of | cancer cells. |
| ing an active immune response in its attack on | cancer cells. |
| y linking T-lymphocytes and macrophages to the | cancer cells. |
| have been identified in different tissues and | cancer cells. |
| as been known to inhibit aerobic glycolysis in | cancer cells. |
| s by inhibiting the telomerase activity of the | cancer cells. |
| However, it is reduced or absent from most | cancer cells. |
| are glycoproteins found on the surface of many | cancer cells. |
| c function in "bookmarking" of target genes in | cancer cells. |
| pears to counteract some of the adaptations of | cancer cells. |
| ein which is implicated in the rapid growth of | cancer cells. |
| nificantly lower levels in non-small cell lung | cancer cells. |
| d chitosan, has been shown to transfect breast | cancer cells; with increased degree of trimethylation |
| Cancer Center | |
| The South Pasadena | Cancer Center specializes in many forms of cancer, in |
| It is also the main women's | cancer center in Illinois. |
| Joan Karnell | Cancer Center |
| The Sylvester Comprehensive | Cancer Center treats 3,000 newly-diagnosed cancer pat |
| M. D. Anderson | Cancer Center Orlando is a cancer treatment facility |
| diation Oncology Department at the Dana-Farber | Cancer Center and a professor of Radiation Oncology a |
| Holycross | Cancer Center (Poland, Kielce) Pathomorphology Depart |
| In December 1971, the Duke Comprehensive | Cancer Center was established under the National Canc |
| She contributed to the Moffitt | Cancer Center by donating "True Hearts" jewelry made |
| culty appointments at Memorial Sloan-Kettering | Cancer Center and Yale University. |
| nce at The University of Chicago Comprehensive | Cancer Center |
| Edwards Comprehensive | Cancer Center |
| entored by Cullen at the Jonsson Comprehensive | Cancer Center at the University of California at Los |
| H. Lee Moffitt | Cancer Center & Research Institute, a four-story, 101 |
| f the University of Pennsylvania Comprehensive | Cancer Center and Institute for Human Gene Therapy. |
| The South Pasadena | Cancer Center is owned and operated by California Can |
| The Massey | Cancer Center in Richmond, Virginia is the charitable |
| In 1973, the USC Norris Comprehensive | Cancer Center was established as one of the original |
| r Board of Overseers, Memorial Sloan Kettering | Cancer Center Board of Overseers, Kravis Leadership I |
| onsors a music program at the Texas Children's | Cancer Center in Houston, Texas. |
| tdoctoral training at Memorial Sloan-Kettering | Cancer Center prior to starting an independent resear |
| The MD Anderson | Cancer Center bought the building in 1974. |
| The Dana-Farber/Harvard | Cancer Center is the largest National Cancer Institut |
| Established in 1991, the M. D. Anderson | Cancer Center Orlando is the first and largest affili |
| M. D. Anderson | Cancer Center Orlando is one of the largest clinical |
| ospital Boston, Massachusetts General Hospital | Cancer Center and Joslin Diabetes Center, offer great |
| UNC Lineberger Comprehensive | Cancer Center is a matrix cancer center at one of the |
| al postdoctoral fellows while at the Fox Chase | Cancer Center in Philadelphia, where the groundbreaki |
| ogy in the United States at the M. D. Anderson | Cancer Center in Houston. |
| Saskatchewan Stroke Research Centre Saskatoon | Cancer Center Research Unit |
| lence in breast and GI cancers, a DoD Prostate | Cancer Center of Excellence, and a recently awarded C |
| nal Kidney Foundation.; and partnering with KU | Cancer Center to raise awareness of esophageal cancer |
| a consultant for the Memorial Sloan-Kettering | Cancer Center in New York for almost a decade prior t |
| The South Pasadena | Cancer Center is a 22,000-square-foot (2,000 m2) stat |
| has donated over $1.7 million to M.D. Anderson | Cancer Center and the American Cancer Society. |
| Emily Couric Clinical | Cancer Center at the University of Virginia Health Sy |
| Malabar | Cancer Center (MCC) established at Kodiyeri near Thal |
| for the Jimmy Everest Children's | Cancer Center at the University of Oklahoma Health Sc |
| f the largest employers in South Pasadena, the | Cancer Center is staffed with California Cancer Speci |
| The primary goal of the | Cancer Center is to encourage and promote collaborati |
| home to the Moses Cone Health System Regional | Cancer Center (designated as a Community Hospital Com |
| gy Department, Scientific Director of the Yale | Cancer Center and a member of the Ludwig Institute fo |
| itution in the United States, and the Hollings | Cancer Center is a National Cancer Institute distingu |
| The | cancer center contains an 8,000-square-foot (740 m2) |
| He was a researcher at the Fox Chase | Cancer Center in Philadelphia (There he worked alongs |
| The USC Norris Comprehensive | Cancer Center is a leader in cancer research, with ne |
| The region's only comprehensive | cancer center offers access to internationally recogn |
| logy at the University of Texas M. D. Anderson | Cancer Center in Houston, Texas and holds the M. G. a |
| he existing units and programs, the Chulabhorn | Cancer Center and Chulabhorn Graduate School are sche |
| The physicians of the | Cancer Center provide inpatient and surgical services |
| Palmetto Health South Carolina | Cancer Center (Cancer Center, Relocated to 11th Floor |
| The James | Cancer Center expanded its efforts in basic research |
| ichi Prefectural Shiroyama Hospital, and Aichi | Cancer Center Hospital. |
| ilion, which features the newly expanded Batte | Cancer Center and Cannon Heart Center. |
| Cancer Center faculty also direct the CDC coordinatin | |
| Molecular Biology at Memorial Sloan-Kettering | Cancer Center in New York. |
| USC Norris Comprehensive | Cancer Center has held this designation since 1973, w |
| Milford Regional opened a new $25-million | cancer center in January of 2007. |
| ia University and the Memorial Sloan-Kettering | Cancer Center in New York City. |
| The St. Vincent's Mary Virginia Terry | Cancer Center is a $20 million, 15,000-square-foot (1 |
| Services offered at the Finney | Cancer Center include chemotherapy and radiation ther |
| The reason for the creation of the | Cancer Center was two-fold: first, the population car |
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