「cytokines」の共起表現(1語右で並び替え) - Weblio英語共起表現検索


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Weblio 辞書 > 英和辞典・和英辞典 > cytokinesの意味・解説 > cytokinesに関連した共起表現

「cytokines」の共起表現一覧(1語右で並び替え)

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In addition, cytokines activate those cells, stimulating them to pr
actors, resulting in the production of other cytokines, an increase in the number of surface recept
cytokines and acute phase proteins, leading to inflamm
us also prevents the release of inflammatory cytokines and mediators from mast cells.
ountering antigens, they immediately release cytokines and cause killing of infected target cells.
receptors for the majority of hematopoietic cytokines and for cytokines that affect other systems,
fected organs die, leading to the release of cytokines and activation of both coagulation and fibri
ion and proteolytic ectodomain processing of cytokines and adhesion molecules.
ng cascade controlling cellular responses to cytokines and stress.
ory role, as knockouts have higher levels of cytokines and are more susceptible to Th2-meditated in
other inflammatory cells through release of cytokines and chemokines induced by ligation of specif
ndothelial cells to secrete pro-inflammatory cytokines and Nitric oxide that lead to "endotoxic sho
e 705 and serine 727, in response to various cytokines and growth factors including interferons, ep
may inhibit the release of preformed T cell cytokines and mediators involved in asthma.
ses include the induction of proinflammatory cytokines, arachidonate mobilization, protein phosphor
The CC cytokines are proteins characterized by two adjacent c
These cytokines are used as therapeutics in many diseases su
matory responses, mediated by the release of cytokines, are involved in the expression of the disea
enes clustered on the p-arm of chromosome 9. Cytokines are a family of secreted proteins involved i
The expression of all three cytokines can be induced by viral infection.
Cytokines can be classified as proteins, peptides, or
Oversecretion of cytokines can trigger a dangerous syndrome known as a
se of a diverse variety of membrane-anchored cytokines, cell adhesion molecules, receptors, ligands
cules, interleukins, mucins, growth factors, cytokines, chemokines, and more.
hain, whose γ-chain (common to several other cytokines) deficiency is directly responsible for the
nfections appear to be stimulated by certain cytokines, e.g., interleukin-1.
as oxidative stress, hypoxia, heavy metals, cytokines, etc. Heme oxygenase 2 (HO-2) is a constitut
n by inhibiting the synthesis and release of cytokines from T-cells.
response to a variety of stimuli, including cytokines, growth factors, stress, and bacterial and v
Adverse effects of cytokines have been linked to many disease states and
ch as oxidized LDL-cholesterol, inflammatory cytokines, hyperhomocysteinemia, hyperglycemia and inf
ion of CXCL16 is induced by the inflammatory cytokines IFN-gamma and TNF-alpha.
ulated in liver cells by the proinflammatory cytokines IL-1, IL-6, and TNF-α.
rve cells, including quinolinic acid and the cytokines IL-6 and TNF-alpha, which can produce fatigu
later shown to correlate with high levels of cytokines in infected humans.
otein kinase and failed to induce downstream cytokines in response to any of the known ligands of T
mon-γ chain receptors is shared with various cytokines, including interleukin-2, -4, -9, and -15.
shown to interact with three closely related cytokines, including interleukin 28A (IL28A), interleu
or-chain that is common to a large family of cytokines, including IL-6, IL-11, IL-27, IL-31, oncost
is the common signal transducer for several cytokines including leukemia inhibitory factor(LIF), c
m myeloid precursor cells in response to the cytokines interleukin 3 (IL-3), interleukin 5 (IL-5),
g stimulation of cells with the inflammatory cytokines interleukin-1 or tumor necrosis factor-alpha
et adhesion) and become activated, releasing cytokines into the area around the injury.
IL-1α and -β are pro-inflammatory cytokines involved in immune defense against infection
xed signal transduction pathways mediated by cytokines, it has been proposed that cytokines and neu
cells that belongs to a family inflammatory cytokines known as chemokines.
of inhibiting synthesis of pro-inflammatory cytokines like IFN-γ, IL-2, IL-3, TNFα and GM-CSF made
resses including osmotic shock, inflammatory cytokines, lipopolysaccharides (LPS), Ultraviolet ligh
It down-regulates the expression of Th1 cytokines, MHC class II antigens, and costimulatory mo
ctious agents, and mediate the production of cytokines necessary for the development of effective i
initiate signalling to induce production of cytokines necessary for the innate immunity and subseq
multiple levels through hormones, mitogens, cytokines, nutrients, etc.
Cytokines of the IL6/GCSF/MGF family are glycoproteins
s p38 MAP kinase in response to inflammatory cytokines or environmental stress.
olic effects have been discovered, including cytokines, orexin (also known as hypocretin), and mela
Cytokines participating in this include tumor necrosis
ndins, growth factors, calcium, inflammatory cytokines, peptide hormones, phorbol esters, and neuro
His research has focused on cytokines, signal transduction pathways and the regula
hen the immune system is fighting pathogens, cytokines signal immune cells such as T-cells and macr
Other targets include genes coding for cytokines such as IL-6, IL-4, IL-5, and TNF-alpha.
y reactions that result in the production of cytokines such as type-I interferon and IL-12.
ease is often stimulated by pro-inflammatory cytokines such as interleukin 1. Inflammatory chemokin
s lipopolysaccharide (LPS), and inflammatory cytokines such as tumor necrosis factor and interferon
t, enhance the production of proinflammatory cytokines, suppress anti-inflammatory mediators and re
n K expression is stimulated by inflammatory cytokines that are released after tissue injury.
SP can also induce the cytokines that are capable of inducing NK-1 transcript
Lymphokines are a subset of cytokines that are produced by a type of immune cell k
can be induced during an immune response, by cytokines that stimulate cells such as fibroblasts and
e immune system to release a large amount of cytokines that lead to significant inflammation.
er protein combines the binding site for two cytokines that regulate maturation, function, and surv
nd on activated T cells and is stimulated by cytokines that promote Th1 cells development and inhib
esistance to interferon and other anti-viral cytokines that is attributed to a single amino acid ch
re are many interactions of IL-1α with other cytokines, the most consistent and most clinically rel
ponse to cellular stress and proinflammatory cytokines, this kinase is activated through its phosph
y diseases, and, recently, tissue protective cytokines to safely treat devastating diseases and inj
at are responsive to stress stimuli, such as cytokines, ultraviolet irradiation, heat shock, and os
The induction of cytokines via toll-like receptor 7 (TLR7) has also bee
different stimuli, including growth factors, cytokines, virus infection, ligands for heterotrimeric
irus induces high levels of chemicals called cytokines when it infects a type of white blood cell.
onic peptides, growth factors, and cytotoxic cytokines, which may act in synergy to amplify cell da
rface of cells that recognize and respond to cytokines with four α-helical strands.
                                                                                                   


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