「cytokines」の共起表現一覧(1語右で並び替え)
該当件数 : 81件
In addition, | cytokines activate those cells, stimulating them to pr |
actors, resulting in the production of other | cytokines, an increase in the number of surface recept |
cytokines and acute phase proteins, leading to inflamm | |
us also prevents the release of inflammatory | cytokines and mediators from mast cells. |
ountering antigens, they immediately release | cytokines and cause killing of infected target cells. |
receptors for the majority of hematopoietic | cytokines and for cytokines that affect other systems, |
fected organs die, leading to the release of | cytokines and activation of both coagulation and fibri |
ion and proteolytic ectodomain processing of | cytokines and adhesion molecules. |
ng cascade controlling cellular responses to | cytokines and stress. |
ory role, as knockouts have higher levels of | cytokines and are more susceptible to Th2-meditated in |
other inflammatory cells through release of | cytokines and chemokines induced by ligation of specif |
ndothelial cells to secrete pro-inflammatory | cytokines and Nitric oxide that lead to "endotoxic sho |
e 705 and serine 727, in response to various | cytokines and growth factors including interferons, ep |
may inhibit the release of preformed T cell | cytokines and mediators involved in asthma. |
ses include the induction of proinflammatory | cytokines, arachidonate mobilization, protein phosphor |
The CC | cytokines are proteins characterized by two adjacent c |
These | cytokines are used as therapeutics in many diseases su |
matory responses, mediated by the release of | cytokines, are involved in the expression of the disea |
enes clustered on the p-arm of chromosome 9. | Cytokines are a family of secreted proteins involved i |
The expression of all three | cytokines can be induced by viral infection. |
Cytokines can be classified as proteins, peptides, or | |
Oversecretion of | cytokines can trigger a dangerous syndrome known as a |
se of a diverse variety of membrane-anchored | cytokines, cell adhesion molecules, receptors, ligands |
cules, interleukins, mucins, growth factors, | cytokines, chemokines, and more. |
hain, whose γ-chain (common to several other | cytokines) deficiency is directly responsible for the |
nfections appear to be stimulated by certain | cytokines, e.g., interleukin-1. |
as oxidative stress, hypoxia, heavy metals, | cytokines, etc. Heme oxygenase 2 (HO-2) is a constitut |
n by inhibiting the synthesis and release of | cytokines from T-cells. |
response to a variety of stimuli, including | cytokines, growth factors, stress, and bacterial and v |
Adverse effects of | cytokines have been linked to many disease states and |
ch as oxidized LDL-cholesterol, inflammatory | cytokines, hyperhomocysteinemia, hyperglycemia and inf |
ion of CXCL16 is induced by the inflammatory | cytokines IFN-gamma and TNF-alpha. |
ulated in liver cells by the proinflammatory | cytokines IL-1, IL-6, and TNF-α. |
rve cells, including quinolinic acid and the | cytokines IL-6 and TNF-alpha, which can produce fatigu |
later shown to correlate with high levels of | cytokines in infected humans. |
otein kinase and failed to induce downstream | cytokines in response to any of the known ligands of T |
mon-γ chain receptors is shared with various | cytokines, including interleukin-2, -4, -9, and -15. |
shown to interact with three closely related | cytokines, including interleukin 28A (IL28A), interleu |
or-chain that is common to a large family of | cytokines, including IL-6, IL-11, IL-27, IL-31, oncost |
is the common signal transducer for several | cytokines including leukemia inhibitory factor(LIF), c |
m myeloid precursor cells in response to the | cytokines interleukin 3 (IL-3), interleukin 5 (IL-5), |
g stimulation of cells with the inflammatory | cytokines interleukin-1 or tumor necrosis factor-alpha |
et adhesion) and become activated, releasing | cytokines into the area around the injury. |
IL-1α and -β are pro-inflammatory | cytokines involved in immune defense against infection |
xed signal transduction pathways mediated by | cytokines, it has been proposed that cytokines and neu |
cells that belongs to a family inflammatory | cytokines known as chemokines. |
of inhibiting synthesis of pro-inflammatory | cytokines like IFN-γ, IL-2, IL-3, TNFα and GM-CSF made |
resses including osmotic shock, inflammatory | cytokines, lipopolysaccharides (LPS), Ultraviolet ligh |
It down-regulates the expression of Th1 | cytokines, MHC class II antigens, and costimulatory mo |
ctious agents, and mediate the production of | cytokines necessary for the development of effective i |
initiate signalling to induce production of | cytokines necessary for the innate immunity and subseq |
multiple levels through hormones, mitogens, | cytokines, nutrients, etc. |
Cytokines of the IL6/GCSF/MGF family are glycoproteins | |
s p38 MAP kinase in response to inflammatory | cytokines or environmental stress. |
olic effects have been discovered, including | cytokines, orexin (also known as hypocretin), and mela |
Cytokines participating in this include tumor necrosis | |
ndins, growth factors, calcium, inflammatory | cytokines, peptide hormones, phorbol esters, and neuro |
His research has focused on | cytokines, signal transduction pathways and the regula |
hen the immune system is fighting pathogens, | cytokines signal immune cells such as T-cells and macr |
Other targets include genes coding for | cytokines such as IL-6, IL-4, IL-5, and TNF-alpha. |
y reactions that result in the production of | cytokines such as type-I interferon and IL-12. |
ease is often stimulated by pro-inflammatory | cytokines such as interleukin 1. Inflammatory chemokin |
s lipopolysaccharide (LPS), and inflammatory | cytokines such as tumor necrosis factor and interferon |
t, enhance the production of proinflammatory | cytokines, suppress anti-inflammatory mediators and re |
n K expression is stimulated by inflammatory | cytokines that are released after tissue injury. |
SP can also induce the | cytokines that are capable of inducing NK-1 transcript |
Lymphokines are a subset of | cytokines that are produced by a type of immune cell k |
can be induced during an immune response, by | cytokines that stimulate cells such as fibroblasts and |
e immune system to release a large amount of | cytokines that lead to significant inflammation. |
er protein combines the binding site for two | cytokines that regulate maturation, function, and surv |
nd on activated T cells and is stimulated by | cytokines that promote Th1 cells development and inhib |
esistance to interferon and other anti-viral | cytokines that is attributed to a single amino acid ch |
re are many interactions of IL-1α with other | cytokines, the most consistent and most clinically rel |
ponse to cellular stress and proinflammatory | cytokines, this kinase is activated through its phosph |
y diseases, and, recently, tissue protective | cytokines to safely treat devastating diseases and inj |
at are responsive to stress stimuli, such as | cytokines, ultraviolet irradiation, heat shock, and os |
The induction of | cytokines via toll-like receptor 7 (TLR7) has also bee |
different stimuli, including growth factors, | cytokines, virus infection, ligands for heterotrimeric |
irus induces high levels of chemicals called | cytokines when it infects a type of white blood cell. |
onic peptides, growth factors, and cytotoxic | cytokines, which may act in synergy to amplify cell da |
rface of cells that recognize and respond to | cytokines with four α-helical strands. |
こんにちは ゲスト さん
ログイン |
Weblio会員(無料)になると 検索履歴を保存できる! 語彙力診断の実施回数増加! |
こんにちは ゲスト さん
ログイン |
Weblio会員(無料)になると 検索履歴を保存できる! 語彙力診断の実施回数増加! |