「endothelial」の共起表現一覧(1語右で並び替え)
該当件数 : 142件
In patients with hypertension, | endothelial activation and damage also lead to changes |
ary to confer high binding affinity for the | endothelial adhesion molecules. |
prognosis (Nakamura et al.): these include | endothelial and autonomic dysfunction (Gori et al.). |
mily of selectins that includes E-selectin ( | endothelial) and L-selectin (leukocyte). |
tion, but it seems to involve inhibition of | endothelial cell migration, proliferation and induction |
a similar function, but is expressed on the | endothelial cell surface within minutes as it is stored |
in "rolling" of the white blood cell on the | endothelial cell surface followed by stable adhesion an |
growth, and the induction of tumor cell and | endothelial cell apoptosis. |
ivity at low concentrations where increased | endothelial cell (EC) migration occurs. |
e of cell-to-cell adhesion in epithelial or | endothelial cell sheets, forming continuous seals aroun |
dies have shown that AMG 706 inhibits human | endothelial cell proliferation induced by VEGF, but not |
many proteins, especially to angiomotin and | endothelial cell surface ATP synthase but also integrin |
In the | endothelial cell lining of blood vessels, endothelial N |
CTGF can promote | endothelial cell growth, migration, adhesion and surviv |
ossibly as a result of its ability to block | endothelial cell chemotaxis. |
In vitro, VEGF has been shown to stimulate | endothelial cell mitogenesis and cell migration. |
development and plays an important role in | endothelial cell biology. |
of T-cells, and makes up a large portion of | endothelial cell intercellular junctions. |
eakdown of the blood-retinal barrier in the | endothelial cell, resulting in leakage of blood product |
l growth regulators, blood platelet-derived | endothelial cell, growth factors, blood platelet-derive |
ys, but a decrease in levels of circulating | endothelial cells was also found . |
n inhibits adhesion of human umbilical vein | endothelial cells (HUVECs) to vitronectin and thus supp |
ane may require a corneal transplant as the | endothelial cells depend on it for support and cannot r |
nesis by promoting migration of neighboring | endothelial cells towards the tumor. |
Ligands present on | endothelial cells will bind to leukocyte expressing L-s |
of the central nervous system as well as on | endothelial cells involved in angiogenesis or angiostas |
reservoir of growth factors is removed the | endothelial cells do not survive, and undergo caspases |
when a cancer cell adheres to the | endothelial cells of a blood vessel it up-regulates the |
he expression of this receptor in lymphatic | endothelial cells and overexpression in vascular tumors |
f inducible nitric oxide synthase (iNOS) in | endothelial cells and cGMP-dependent protein kinase 1α/ |
vation of primed neutrophils and react with | endothelial cells expressing PR3. |
in turn causes release of nitric oxide from | endothelial cells in the trabecular arteries. |
an RNA element secreted by fibroblasts and | endothelial cells in response to the inflammatory media |
Endothelial cells produce nitric oxide (NO) which cause | |
regulated in some aggressive tumors and the | endothelial cells and pericytes of some tumor blood ves |
y model), astrocytes (neurotoxicity model), | endothelial cells (vascular toxicity model), and airway |
ues including heart, lung and ovary, and in | endothelial cells that have been stimulated with the cy |
hions are thought to arise from a subset of | endothelial cells that undergo epithelial to mesenchyma |
A. The | endothelial cells of the glomerulus; 1. pore (fenestra) |
These cell types include monocytes, | endothelial cells and fibroblasts. |
APC has anti-inflammatory effects on | endothelial cells and leukocytes. |
a cell adhesion molecule expressed only on | endothelial cells activated by cytokines. |
When | endothelial cells are activated by molecules such as hi |
he lymphatic capillary, attaching lymphatic | endothelial cells to surrounding tissues. |
The ends of | endothelial cells that make up the wall of a lymphatic |
achment of monocyte derived immune cells to | endothelial cells is also enhanced by TIE1 expression. |
Instead, vascular | endothelial cells will use L-arginine to make nitric ox |
APC affects | endothelial cells by inhibiting inflammatory mediator r |
dition, CXCR4 can also be found on vascular | endothelial cells and neuronal/nerve cells. |
any cell types, including epithelial cells, | endothelial cells and leukocytes. |
It was first shown to be found in | endothelial cells in 1989. |
Increasing tetrahydrobiopterin in | endothelial cells by augmenting the levels of the biosy |
lsidomine and acts by releasing NO from the | endothelial cells nonenzymatically. |
ed primarily to demonstrate the presence of | endothelial cells in histological tissue sections. |
tercellular connection between two adjacent | endothelial cells to fail and allows the cancer cell to |
dendritic cells, hepatocytes, neurons, and | endothelial cells but is not produced by lymphocytes. |
glycoprotein found on white blood cells and | endothelial cells that binds to P-selectin (P stands fo |
acting with P-selectin and/or E-selectin on | endothelial cells and adherent platelets. |
ion by endostatin inhibits the migration of | endothelial cells through disruption of cell-matrix adh |
Malignant | endothelial cells also commonly retain the antigen, so |
see "Intermediate-sized filaments of human | endothelial cells" by Franke, Schmid, Osborn and Weber) |
creted by macrophages, T cells, mast cells, | endothelial cells, and fibroblasts. |
protein that appears to be associated with | endothelial cells, and has been implicated to function |
entral nervous system, intestinal myocytes, | endothelial cells, and hepatocytes. |
nd acts primarily upon epithelial cells and | endothelial cells, but also acts on haemopoietic progen |
molecule (CAM) on the surfaces of activated | endothelial cells, which line the inner surface of bloo |
-8 there is a relative wide range of cells ( | endothelial cells, macrophages, mast cells, keratinocyt |
dy showed a decrease in VEGF-C, circulating | endothelial cells, and CD146 and CD34/133 counts, and a |
itic cells, promotion of T cell adhesion to | endothelial cells, antitumor activity, and inhibition o |
response mediators in leukocytes as well as | endothelial cells, by reducing cytokine response, and m |
muscle cells, liver cells (and also in the | endothelial cells, Kupffer cells and stellate cells of |
ues, mainly in the lung, including vascular | endothelial cells, epithelial kidney cells, and testicu |
es, T-cell lines, activated T lymphoblasts, | endothelial cells, and activated platelets (Lin et al., |
rs in the cells that make up blood vessels ( | endothelial cells, smooth muscle cells and pericytes) b |
cal adhesion and invasion of epithelial and | endothelial cells, mediating attachment to host cells b |
-haematopoietic cells (smooth muscle cells, | endothelial cells, and epithelial cells). |
tes, eosinophils, mast cells and basophils, | endothelial cells, platelets, monocytes and myeloid cel |
comprises a single layer of non-fenestrated | endothelial cells, which have tight junctions. |
It is also synthesized by | endothelial cells, which store IL-8 in their storage ve |
CD-31 is normally found on | endothelial cells, platelets, macrophages and Kupffer c |
In unactivated | endothelial cells, it is stored in granules called Weib |
ll surface protein expressed exclusively in | endothelial cells, however it has also been shown to be |
ding neutrophils, basophils, platelets, and | endothelial cells. |
odes COX-1, which regulates angiogenesis in | endothelial cells. |
been found to accelerate differentiation of | endothelial cells. |
ograph of a liver sinusoid with fenestrated | endothelial cells. |
arated from the lumen of the capillaries by | endothelial cells. |
ase-3-dependent apoptosis of cultured human | endothelial cells. |
kinase-induced migration of fibroblast and | endothelial cells. |
inhibits the production of interleukin-6 in | endothelial cells. |
as glycosylphosphatidyl anchored enzymes on | endothelial cells. |
ificantly affect 12% of genes used by human | endothelial cells. |
n fibroblasts, leukocytes, and blood vessel | endothelial cells. |
macrophages, cardiomyocytes, adipocytes and | endothelial cells. |
aster of Surgery (1990) for work on corneal | endothelial changes after cataract surgery and a Doctor |
significantly higher postoperative corneal | endothelial counts in the Healon eyes as opposed to the |
Endothelial dysfunction leads to the leakage of fluid f | |
protection from | endothelial dysfunction (important facet of atheroscler |
These alterations together lead to both | endothelial dysfunction and coagulation disorder. |
Oxide can lead to vascular damage, such as | endothelial dysfunction and vascular inflammation. |
Alterations in | endothelial function are a reliable indicator of target |
AZD2171, is a potent inhibitor of vascular | endothelial growth factor (VEGF) receptor tyrosine kina |
nzyme involved in the induction of vascular | endothelial growth factor (VEGF)-stimulated neo-angioge |
as ZD6474, is an antagonist of the vascular | endothelial growth factor receptor (VEGFR) and the epid |
tor tyrosine kinase) also known as vascular | endothelial growth factor receptor 2 (VEGFR-2) is a VEG |
se current research, by decreasing vascular | endothelial growth factor production and receptor phosp |
It selectively targets vascular | endothelial growth factor receptors 1, 2 and 3, platele |
Vascular | endothelial growth factor (VEGF) is an important signal |
It functions as an inhibitor of vascular | endothelial growth factor. |
vasculogenesis, angiogenesis, and vascular | endothelial growth factor (VEGF). |
GF has been shown to interact with Vascular | endothelial growth factor A. |
S1 has been shown to interact with Vascular | endothelial growth factor A. |
tibody (IgG1) directed against the Vascular | Endothelial Growth Factor Receptor 2 (VEGFR2). |
or, the MuSK protein receptor, the Vascular | Endothelial Growth Factor (or VEGF) receptor, etc. |
antagonist blocking the binding of vascular | endothelial growth factor (VEGF) to its receptor (VEGF |
ical research into the pathways of improved | endothelial health via downregulation of oxidative LDL. |
ns allows passive spread of agonist-induced | endothelial hyperpolarization through the vessel wall. |
K+ ions may themselves initiate a conducted | endothelial hyperpolarization, thus suggesting that ele |
lammatory effect and may play a role in the | endothelial inflammatory diseases such as atheroscleros |
Berberine hence protects against | endothelial injury and enhances the endothelium-depende |
oper substance, also called stroma, and the | endothelial layer of the cornea. |
The | endothelial layer is located at the posterior of the co |
membrane, as the basement membrane for the | endothelial layer, is secreted by the single layer of s |
Thus a spiralling effect occurs with high | endothelial LDL levels causing greater ADMA values, whi |
this they lose all their coats except their | endothelial lining, which is continuous with a layer of |
tic lipase, hepatic lipase, gastric lipase, | endothelial lipase, and lipoprotein lipase |
receptor and non-receptor tyrosine kinases, | endothelial nitric oxide synthase (eNOS), and small GTP |
It induces | endothelial nitric oxide synthesis by making endothelia |
en Rosen, where she studied the activity of | endothelial oligosaccharides in promoting cell adhesion |
Iridocorneal | endothelial or ICE syndrome is a spectrum of conditions |
antly found in ectoderm derived tissues and | endothelial organ lining, i.e. skin and internal organs |
A wide range of pathologic stimuli induce | endothelial oxidative stress such as oxidized LDL-chole |
Endothelial PAS domain-containing protein 1 is a protei | |
arthritis in response to TNF-α, increasing | endothelial permeability and swelling and also stimulat |
ts in vasoconstriction due to its action on | endothelial postsynaptic α2 receptors; systemic applica |
3 is expressed in hematopoietic stem cells, | endothelial progenitor cells, glioblastoma, neuronal an |
010) has shown that ID1 can be used to mark | Endothelial Progentior Cells which are critical to tumo |
Endothelial protein C receptor (EPCR) also known as act | |
e tying this effect to neuronal rather than | endothelial reactions. |
ralization of heparin-like molecules on the | endothelial surface of blood vessels, thereby inhibitin |
, which are now able to tightly bind to the | endothelial surface and begin making their way into the |
lammation regulation: Glycocalyx coating on | endothelial walls in blood vessels prevents leukocytes |
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