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Weblio 辞書 > 英和辞典・和英辞典 > endothelialの意味・解説 > endothelialに関連した共起表現

「endothelial」の共起表現一覧(1語右で並び替え)

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In patients with hypertension, endothelial activation and damage also lead to changes
ary to confer high binding affinity for the endothelial adhesion molecules.
prognosis (Nakamura et al.): these include endothelial and autonomic dysfunction (Gori et al.).
mily of selectins that includes E-selectin ( endothelial) and L-selectin (leukocyte).
tion, but it seems to involve inhibition of endothelial cell migration, proliferation and induction
a similar function, but is expressed on the endothelial cell surface within minutes as it is stored
in "rolling" of the white blood cell on the endothelial cell surface followed by stable adhesion an
growth, and the induction of tumor cell and endothelial cell apoptosis.
ivity at low concentrations where increased endothelial cell (EC) migration occurs.
e of cell-to-cell adhesion in epithelial or endothelial cell sheets, forming continuous seals aroun
dies have shown that AMG 706 inhibits human endothelial cell proliferation induced by VEGF, but not
many proteins, especially to angiomotin and endothelial cell surface ATP synthase but also integrin
In the endothelial cell lining of blood vessels, endothelial N
CTGF can promote endothelial cell growth, migration, adhesion and surviv
ossibly as a result of its ability to block endothelial cell chemotaxis.
In vitro, VEGF has been shown to stimulate endothelial cell mitogenesis and cell migration.
development and plays an important role in endothelial cell biology.
of T-cells, and makes up a large portion of endothelial cell intercellular junctions.
eakdown of the blood-retinal barrier in the endothelial cell, resulting in leakage of blood product
l growth regulators, blood platelet-derived endothelial cell, growth factors, blood platelet-derive
ys, but a decrease in levels of circulating endothelial cells was also found .
n inhibits adhesion of human umbilical vein endothelial cells (HUVECs) to vitronectin and thus supp
ane may require a corneal transplant as the endothelial cells depend on it for support and cannot r
nesis by promoting migration of neighboring endothelial cells towards the tumor.
Ligands present on endothelial cells will bind to leukocyte expressing L-s
of the central nervous system as well as on endothelial cells involved in angiogenesis or angiostas
reservoir of growth factors is removed the endothelial cells do not survive, and undergo caspases
when a cancer cell adheres to the endothelial cells of a blood vessel it up-regulates the
he expression of this receptor in lymphatic endothelial cells and overexpression in vascular tumors
f inducible nitric oxide synthase (iNOS) in endothelial cells and cGMP-dependent protein kinase 1α/
vation of primed neutrophils and react with endothelial cells expressing PR3.
in turn causes release of nitric oxide from endothelial cells in the trabecular arteries.
an RNA element secreted by fibroblasts and endothelial cells in response to the inflammatory media
Endothelial cells produce nitric oxide (NO) which cause
regulated in some aggressive tumors and the endothelial cells and pericytes of some tumor blood ves
y model), astrocytes (neurotoxicity model), endothelial cells (vascular toxicity model), and airway
ues including heart, lung and ovary, and in endothelial cells that have been stimulated with the cy
hions are thought to arise from a subset of endothelial cells that undergo epithelial to mesenchyma
A. The endothelial cells of the glomerulus; 1. pore (fenestra)
These cell types include monocytes, endothelial cells and fibroblasts.
APC has anti-inflammatory effects on endothelial cells and leukocytes.
a cell adhesion molecule expressed only on endothelial cells activated by cytokines.
When endothelial cells are activated by molecules such as hi
he lymphatic capillary, attaching lymphatic endothelial cells to surrounding tissues.
The ends of endothelial cells that make up the wall of a lymphatic
achment of monocyte derived immune cells to endothelial cells is also enhanced by TIE1 expression.
Instead, vascular endothelial cells will use L-arginine to make nitric ox
APC affects endothelial cells by inhibiting inflammatory mediator r
dition, CXCR4 can also be found on vascular endothelial cells and neuronal/nerve cells.
any cell types, including epithelial cells, endothelial cells and leukocytes.
It was first shown to be found in endothelial cells in 1989.
Increasing tetrahydrobiopterin in endothelial cells by augmenting the levels of the biosy
lsidomine and acts by releasing NO from the endothelial cells nonenzymatically.
ed primarily to demonstrate the presence of endothelial cells in histological tissue sections.
tercellular connection between two adjacent endothelial cells to fail and allows the cancer cell to
dendritic cells, hepatocytes, neurons, and endothelial cells but is not produced by lymphocytes.
glycoprotein found on white blood cells and endothelial cells that binds to P-selectin (P stands fo
acting with P-selectin and/or E-selectin on endothelial cells and adherent platelets.
ion by endostatin inhibits the migration of endothelial cells through disruption of cell-matrix adh
Malignant endothelial cells also commonly retain the antigen, so
see "Intermediate-sized filaments of human endothelial cells" by Franke, Schmid, Osborn and Weber)
creted by macrophages, T cells, mast cells, endothelial cells, and fibroblasts.
protein that appears to be associated with endothelial cells, and has been implicated to function
entral nervous system, intestinal myocytes, endothelial cells, and hepatocytes.
nd acts primarily upon epithelial cells and endothelial cells, but also acts on haemopoietic progen
molecule (CAM) on the surfaces of activated endothelial cells, which line the inner surface of bloo
-8 there is a relative wide range of cells ( endothelial cells, macrophages, mast cells, keratinocyt
dy showed a decrease in VEGF-C, circulating endothelial cells, and CD146 and CD34/133 counts, and a
itic cells, promotion of T cell adhesion to endothelial cells, antitumor activity, and inhibition o
response mediators in leukocytes as well as endothelial cells, by reducing cytokine response, and m
muscle cells, liver cells (and also in the endothelial cells, Kupffer cells and stellate cells of
ues, mainly in the lung, including vascular endothelial cells, epithelial kidney cells, and testicu
es, T-cell lines, activated T lymphoblasts, endothelial cells, and activated platelets (Lin et al.,
rs in the cells that make up blood vessels ( endothelial cells, smooth muscle cells and pericytes) b
cal adhesion and invasion of epithelial and endothelial cells, mediating attachment to host cells b
-haematopoietic cells (smooth muscle cells, endothelial cells, and epithelial cells).
tes, eosinophils, mast cells and basophils, endothelial cells, platelets, monocytes and myeloid cel
comprises a single layer of non-fenestrated endothelial cells, which have tight junctions.
It is also synthesized by endothelial cells, which store IL-8 in their storage ve
CD-31 is normally found on endothelial cells, platelets, macrophages and Kupffer c
In unactivated endothelial cells, it is stored in granules called Weib
ll surface protein expressed exclusively in endothelial cells, however it has also been shown to be
ding neutrophils, basophils, platelets, and endothelial cells.
odes COX-1, which regulates angiogenesis in endothelial cells.
been found to accelerate differentiation of endothelial cells.
ograph of a liver sinusoid with fenestrated endothelial cells.
arated from the lumen of the capillaries by endothelial cells.
ase-3-dependent apoptosis of cultured human endothelial cells.
kinase-induced migration of fibroblast and endothelial cells.
inhibits the production of interleukin-6 in endothelial cells.
as glycosylphosphatidyl anchored enzymes on endothelial cells.
ificantly affect 12% of genes used by human endothelial cells.
n fibroblasts, leukocytes, and blood vessel endothelial cells.
macrophages, cardiomyocytes, adipocytes and endothelial cells.
aster of Surgery (1990) for work on corneal endothelial changes after cataract surgery and a Doctor
significantly higher postoperative corneal endothelial counts in the Healon eyes as opposed to the
Endothelial dysfunction leads to the leakage of fluid f
protection from endothelial dysfunction (important facet of atheroscler
These alterations together lead to both endothelial dysfunction and coagulation disorder.
Oxide can lead to vascular damage, such as endothelial dysfunction and vascular inflammation.
Alterations in endothelial function are a reliable indicator of target
AZD2171, is a potent inhibitor of vascular endothelial growth factor (VEGF) receptor tyrosine kina
nzyme involved in the induction of vascular endothelial growth factor (VEGF)-stimulated neo-angioge
as ZD6474, is an antagonist of the vascular endothelial growth factor receptor (VEGFR) and the epid
tor tyrosine kinase) also known as vascular endothelial growth factor receptor 2 (VEGFR-2) is a VEG
se current research, by decreasing vascular endothelial growth factor production and receptor phosp
It selectively targets vascular endothelial growth factor receptors 1, 2 and 3, platele
Vascular endothelial growth factor (VEGF) is an important signal
It functions as an inhibitor of vascular endothelial growth factor.
vasculogenesis, angiogenesis, and vascular endothelial growth factor (VEGF).
GF has been shown to interact with Vascular endothelial growth factor A.
S1 has been shown to interact with Vascular endothelial growth factor A.
tibody (IgG1) directed against the Vascular Endothelial Growth Factor Receptor 2 (VEGFR2).
or, the MuSK protein receptor, the Vascular Endothelial Growth Factor (or VEGF) receptor, etc.
antagonist blocking the binding of vascular endothelial growth factor (VEGF) to its receptor (VEGF
ical research into the pathways of improved endothelial health via downregulation of oxidative LDL.
ns allows passive spread of agonist-induced endothelial hyperpolarization through the vessel wall.
K+ ions may themselves initiate a conducted endothelial hyperpolarization, thus suggesting that ele
lammatory effect and may play a role in the endothelial inflammatory diseases such as atheroscleros
Berberine hence protects against endothelial injury and enhances the endothelium-depende
oper substance, also called stroma, and the endothelial layer of the cornea.
The endothelial layer is located at the posterior of the co
membrane, as the basement membrane for the endothelial layer, is secreted by the single layer of s
Thus a spiralling effect occurs with high endothelial LDL levels causing greater ADMA values, whi
this they lose all their coats except their endothelial lining, which is continuous with a layer of
tic lipase, hepatic lipase, gastric lipase, endothelial lipase, and lipoprotein lipase
receptor and non-receptor tyrosine kinases, endothelial nitric oxide synthase (eNOS), and small GTP
It induces endothelial nitric oxide synthesis by making endothelia
en Rosen, where she studied the activity of endothelial oligosaccharides in promoting cell adhesion
Iridocorneal endothelial or ICE syndrome is a spectrum of conditions
antly found in ectoderm derived tissues and endothelial organ lining, i.e. skin and internal organs
A wide range of pathologic stimuli induce endothelial oxidative stress such as oxidized LDL-chole
Endothelial PAS domain-containing protein 1 is a protei
arthritis in response to TNF-α, increasing endothelial permeability and swelling and also stimulat
ts in vasoconstriction due to its action on endothelial postsynaptic α2 receptors; systemic applica
3 is expressed in hematopoietic stem cells, endothelial progenitor cells, glioblastoma, neuronal an
010) has shown that ID1 can be used to mark Endothelial Progentior Cells which are critical to tumo
Endothelial protein C receptor (EPCR) also known as act
e tying this effect to neuronal rather than endothelial reactions.
ralization of heparin-like molecules on the endothelial surface of blood vessels, thereby inhibitin
, which are now able to tightly bind to the endothelial surface and begin making their way into the
lammation regulation: Glycocalyx coating on endothelial walls in blood vessels prevents leukocytes
                                                                                                   


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