「receptors」の共起表現一覧(1語右で並び替え)5ページ目
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rons may interact with alpha-2A and alpha-2C | receptors to inhibit neurally released norepinephrine. |
been proposed to play a role in movement of | receptors to the plasma membrane. |
vator that can interact with nuclear hormone | receptors to enhance their transcriptional activator f |
s its effects through ionotropic [GABA(A/C)] | receptors, to produce fast synaptic inhibition, and me |
ubunit alters the sensitivity of recombinant | receptors to modulatory agents such as pregnanolone. |
at bridges the Fas-receptor, and other death | receptors, to caspase-8 through its death domain to fo |
The renal effects allow the | receptors to change the mean pressure in the system in |
in is involved in the transportation of AMPA | receptors to the synaptic membrane, and the regulation |
The sensitivity of P2X | receptors to ATP is strongly modulated by changes in e |
The Activin type I | receptors transduce signals for a variety of members o |
ligands for heterotrimeric G protein-coupled | receptors, transforming agents, and carcinogens, activ |
chemoeffector gradients involves chemotaxis | receptors, transmembrane (TM) proteins that detect sti |
Activated I1-imidazoline | receptors trigger the hydrolysis of phosphatidylcholin |
of TGFβ superfamily ligands to extracellular | receptors triggers phosphorylation of Smad2 at a Serin |
Activation of central 5-HT1A | receptors triggers the release or inhibition of norepi |
Chemokine | receptors, two of which acting as binding proteins for |
different types of serine (threonine) kinase | receptors: type I receptors of about 50-55 kD and type |
central nervous system effects on muscarinic | receptors, type 4 and 5. |
tegory of organic molecules that includes Fc | receptors, Ulks, Calcineurins, K chips, and urocortins |
ctin ligands and fibrin, but not fibrinogen, | receptors under hemodynamic flow conditions pertinent |
with preferential actions over α2-adrenergic | receptors, underlying its hypotensive rather than hype |
c effects apparently mediated through opioid | receptors, unlike the inactive natural enantiomer (-)- |
over, itopride has no affinity for the 5-HT4 | receptors unlike other benzamides such as cisapride an |
d efficacious agonist at the M1 and δ-opioid | receptors, unlike clozapine as well. |
Interaction of this protein with the | receptors unmasks the N-terminal effector domain(see s |
By activating 5-HT2C | receptors, vabicaserin inhibits dopamine release in th |
nates are innervated by pain and temperature | receptors, via the trigeminal nerve (or, the fifth cra |
aptor protein that forms a complex with many | receptors via its PTB domain. |
Quazepam modulates specific GABAA | receptors via the benzodiazepine site on the GABAA rec |
lly indirectly affected by G protein-coupled | receptors via effector proteins (such as phospholipase |
omodimers or heterodimers with retinoic acid | receptors, vitamin D receptors, thyroid receptors or p |
ribed as 100-fold selective for GluN1/GluN2A | receptors vs. GluN1/GluN2B receptors, more detailed st |
New terminology for NOD-like | receptors was adopted by the Human Genome Organization |
uroleptic because it does not block dopamine | receptors well. |
tint in a detox program, in which his opiate | receptors were cleaned. |
Historically, | receptors were discovered by using ligands to "fish" f |
concentration indicating that the COX enzyme | receptors were already saturated and blocked by the br |
c drugs, which do not act on the D2 dopamine | receptors were thought to have reduced the incidence o |
Hence by definition, these | receptors were not orphans. |
Moreover, 5-HT2B | receptors were recently shown to be overexpressed in h |
agonist, and has been used to identify these | receptors when labeled with tritium. |
Like other type II nuclear | receptors, when activated, it forms a heterodimer with |
es (also known as immunoglobulins) or T cell | receptors where these proteins complement an antigen's |
with the exception of the 5-HT2A and 5-HT2C | receptors where it acts as a partial agonist. |
y norepinephrine (noradrenaline) (adrenergic | receptors), whereas parasympathetic systems are acetyl |
M1, M2, M3, M4 and M5 subtypes of muscarinic | receptors whereas modern antimuscarinic treatments for |
A single antibody molecule has two antigen | receptors, wherefore it contains twelve CDRs. |
ked seven-transmembrane spanning leukotriene | receptors which are highly expressed in inflammation a |
Its inhibition of beta-2 | receptors, which are mainly located in the bronchial s |
has also been shown to colocalize with GABAA | receptors, which serve as ligand-gated ion channels to |
rent types of bacterial 60 kDa transmembrane | receptors, which share similar topology and signalling |
selective, mixed agonist-antagonist at GABAA | receptors, which acts as a partial agonist at the α2 a |
through a site separate from the CB1 and CB2 | receptors, which responds to abnormal cannabidiol, O-1 |
n the mammalian brain where it acts at GABAA | receptors, which are ligand-gated chloride channels. |
the mammalian brain where it acts at GABA-A | receptors, which are ligand-gated chloride channels. |
It has a preference for beta-1 | receptors, which are predominantly located in the hear |
retinoid X receptor (RXR) family of nuclear | receptors which are involved in mediating the antiprol |
d with the indirect function of metabotropic | receptors, which use second messengers. |
I result in unopposed stimulation of the AT2 | receptors, which are, in addition, upregulated. |
through the stimulation of cell surface ACTH | receptors, which are located primarily on adrenocortic |
The | receptors, which are located on liver cells, remove th |
SB-269,970 is used to study the 5-HT7 | receptors which are thought to be involved in the func |
in the mammalian brain where it acts at GABA | receptors, which are ligand-gated chloride channels. |
eceptor antagonist of the OX1 and OX2 orexin | receptors, which was being developed by the pharmaceut |
the mammalian brain where it acts at GABA-A | receptors, which are ligand-gated chloride channels. |
duced through the agonization of cannabinoid | receptors which prevents microglial activation that el |
n is relatively selective for α1a-adrenergic | receptors, which are mainly present in the prostate. |
bits the dimerization of HER2 with other HER | receptors, which is hypothesized to result in slowed t |
novel family of C-type lectin transmembrane | receptors which play a role not only in cell-cell adhe |
e, potent and selective agonist for androgen | receptors which was shown to have anabolic effects in |
CD93 is a C-type lectin transmembrane | receptors which play a role not only in cell-cell adhe |
a much longer-lasting effect than ionotropic | receptors, which open quickly but only remain open for |
isoning by blocking muscarinic acetylcholine | receptors, which would otherwise be overstimulated by |
t role in this kindling phenomenon with AMPA | receptors which are a subtype of glutamate receptors b |
gene is a member of the family of serotonin | receptors, which are G protein coupled receptors that |
Unlike the H1 and H2 | receptors which have primarily peripheral actions, but |
ugs selectively targets type1 benzodiazepine | receptors which results reduced sleep latency in promo |
tein belongs to the subfamily B class of LIR | receptors which contain two or four extracellular immu |
a subunit of the N-methyl-D-aspartate (NMDA) | receptors, which belong to the superfamily of glutamat |
ent focus because glutamate blocks some NMDA | receptors which, on their own, induce schizophrenic be |
are produced through activation of melatonin | receptors, while others are due to its role as a perva |
t studies in mice suggest that the different | receptors, while having certain extent of redundancy, |
RANTES) are working on long-term chemotaxis | receptors, while vasoactive peptides (e.g. |
blocking glutamatergic N-methyl-D-aspartate | receptors, while gamma-aminobutyric acid (GABA) type A |
eptor family is a group of G-protein coupled | receptors whose principal ligand is the protein bradyk |
ns in the TLR family are pattern recognition | receptors whose task is to alert the immune system of |
Type I cytokine | receptors, whose members have certain conserved motifs |
Respectively, these | receptors will increase somatostatin output and decrea |
of deep burn there will be no pain as these | receptors will be burned off. |
It has high affinity for both CB1 and CB2 | receptors, with Ki values of 1.5nM at CB1 and 0.32nM a |
α7 subtype of neural nicotinic acetylcholine | receptors, with a high level of brain penetration and |
MG-3 is a potent agonist at both CB1 and CB2 | receptors with a Ki of 0.32nM at CB1 and 0.52nM at CB2 |
as fairly high affinity for both CB1 and CB2 | receptors, with Ki values of 2.91nM at CB1 and 4.24nM |
BN acts as a weak agonist of the CB1 and CB2 | receptors, with lower affinity in comparison to THC. |
tagonist for the 5-HT2B and 5-HT2C serotonin | receptors, with good selectivity over other serotonin |
is a potent agonist at both the CB1 and CB2 | receptors, with a binding affinity of 0.1nM at CB1 and |
lgesia involves blocking activation of these | receptors with NMDAR antagonists such as ketamine, dex |
ered the first family of mammalian pheromone | receptors with Nobel laureate Richard Axel. |
antagonism at dopamine and serotonin type 2 | receptors, with greater activity at serotonin 5-HT2 re |
laced radiolabelled ketanserin from 5-HT2A/C | receptors with a Ki of 74.5, as compared to a Ki of 80 |
a subtype-selective partial agonist at GABAA | receptors, with highest affinity for the α3 subtype, b |
t has moderate affinity for both CB1 and CB2 | receptors, with Ki values of 8.36nM at CB1 and 7.95nM |
ve agonist for the 5-HT2 family of serotonin | receptors, with highest binding affinity for the 5-HT2 |
It is a partial agonist at CB1 | receptors, with a Ki of 87nM, making it roughly half t |
an agonist at neural nicotinic acetylcholine | receptors with high selectivity for the α4β2 subtype. |
elective agonist for nicotinic acetylcholine | receptors, with twice the potency of nicotine. |
and has modest affinity for both CB1 and CB2 | receptors, with Ki values of 48.3nM at CB1 and 45.5nM |
showed potent antagonism of human muscarinic | receptors, with a long residence time at M3 receptors |
cannabinoid agonist at both the CB1 and CB2 | receptors, with Ki values of 0.46nM at CB1 and 0.69nM |
in belongs to a subfamily of tyrosine kinase | receptors with a homology region to the Dictyostelium |
eptor, since either subunit alone results in | receptors with very low conductance and response ampli |
and kainate families of ionotropic glutamate | receptors, with selectivity for the GluR5 subtype of t |
eceptor antagonist and also inhibits glycine | receptors with similar potency and nicotinic acetylcho |
ghly potent full agonist for the cannabinoid | receptors, with a Ki of 1.8nM at CB1 and 2.2nM at CB2 |
denosine receptor group of G protein-coupled | receptors with adenosine as endogenous ligand. |
agonist of both the CB1 and CB2 cannabinoid | receptors, with a reported binding affinity of 9.00+/- |
oderately potent agonist for the cannabinoid | receptors, with a Ki of 13.5nM at CB1 and 49.5nM at CB |
211, is a mixed agonist-antagonist at opioid | receptors with a similar pharmacological profile to pe |
It has high affinity for these | receptors with dissociation constants of 2.8 nM and 3. |
oximately equal affinity for the CB1 and CB2 | receptors, with a Ki of 6.6nM at CB1 and 6.9nM at CB2. |
It forms one subunit of type I cytokine | receptors within the IL-6 receptor family. |
ester that selectively stimulates muscarinic | receptors without any effect on nicotinic receptors. |
(5-HT2), adrenaline (α1), and histamine (H1) | receptors, without affecting the muscarinic acetylchol |
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