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Weblio 辞書 > 英和辞典・和英辞典 > receptorsの意味・解説 > receptorsに関連した共起表現

「receptors」の共起表現一覧(2語左で並び替え)

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The type A GABA receptors are pentameric chloride channels assembled f
binding and signaling through a TrkC-related receptors called TrkB.
It can act on receptors at pre-ganglionic sites in both the sympathe
es can act in numerous ways, often acting as receptors or ligands (the molecule that activates a re
cell contact, ion conductance, and acting as receptors.
of flunisolide is to activate glucocorticoid receptors.
elatively wide gap (>20 µm) to activate 5-HT receptors located on the dendrites, cell bodies and pr
ere are 11 growth factors that activate ErbB receptors.
Like capsaicin, capsinoids activate TRPV1 receptors, although they are not hot in the mouth.
both capsaicin and capsinoids activate TRPV1 receptors in the same manner.
promotion sites by ligand activated nuclear receptors.
Activated I1-imidazoline receptors trigger the hydrolysis of phosphatidylcholin
gonist is a compound that activates dopamine receptors in the absence of dopamine.
IP3 enters the cytoplasm and activates IP3 receptors on the smooth endoplasmic reticulum (ER), wh
rmone cortisol that activates glucocorticoid receptors.
ediated effects but also activates adenosine receptors, often with opposing (patho-) physiological
ns that serotonin has when it activates 5-HT receptors.
uronal signaling molecule by activating NMDA receptors in the brain.
al selectivity by strongly activating 5-HT1A receptors in the prefrontal cortex while having little
By activating 5-HT2C receptors, vabicaserin inhibits dopamine release in th
periments using constitutively active BMPR1B receptors.
ceptor and reverses constitutive activity of receptors.
ts there are yet more additional cannabinoid receptors.
Adenosine A3 receptors are G protein-coupled receptors that are inv
anchored cytokines, cell adhesion molecules, receptors, ligands, and enzymes.
Adopted orphan receptors in the nuclear receptor group include the fa
localizes with intracellular adrenal steroid receptors and converts cortisol (an active mineralcort
dy produces antibodies against acetylcholine receptors, and thus inhibits signal transmission acros
The renal effects allow the receptors to change the mean pressure in the system in
he rat parotid gland and at alpha adrenergic receptors.
partial agonist action at Alpha-2 adrenergic receptors, and TDIQ has been suggested as a possible d
ular domain of the protein which is also the receptors G protein coupling domain.Therefore editing
tain CCKBR (which respond to gastrin) and M3 receptors (which respond to Ach).
cannabinoid agonist at both the CB1 and CB2 receptors.
rect or indirect activation of CB2 and TRPV1 receptors, and induction of oxidative stress, all cont
as a low affinity for AMPA, kainate and NMDA receptors.
y over the closely related 5-HT2A and 5-HT2B receptors.
onsidered a full agonist at both CB1 and CB2 receptors and has Ki values of 0.58nM and 0.68nM respe
mary actions of steroid hormones and nuclear receptors.
energic, α2-adrenergic, β-adrenergic, and D2 receptors.
nates are innervated by pain and temperature receptors, via the trigeminal nerve (or, the fifth cra
pe I (I and IB) and two type II (II and IIB) receptors.
a selective antagonist at dopamine D2 and D3 receptors.
It has high affinity for both CB1 and CB2 receptors, with Ki values of 1.5nM at CB1 and 0.32nM a
ive in synthesizing shape- and ion-selective receptors, and throughout the 1980s research in the ar
selective agonist for the 5-HT2A and 5-HT2C receptors, but unlike the corresponding benzocyclobute
r binding affinity for the 5-HT1A and 5-HT2A receptors, but is less potent in producing drug-approp
eceptor full agonist at both the CB1 and CB2 receptors.
MG-3 is a potent agonist at both CB1 and CB2 receptors with a Ki of 0.32nM at CB1 and 0.52nM at CB2
, the macrophage expresses more CD40 and TNF receptors on its surface which helps increase the leve
idence for receptor overexpression and spare receptors suggests that the calculation of the net cha
cardioselectivity and blocks both β1 and β2 receptors.
as fairly high affinity for both CB1 and CB2 receptors, with Ki values of 2.91nM at CB1 and 4.24nM
through a site separate from the CB1 and CB2 receptors, which responds to abnormal cannabidiol, O-1
ved to work by blockading dopamine D1 and D2 receptors in the mesocortical and mesolimbic pathways,
ctive full agonist at the dopamine D1 and D5 receptors.
acts as an agonist at the 5-HT1A and 5-HT1B receptors and as an antagonist at the 5-HT2C receptor.
signaling pathways, coregulators and nuclear receptors at a promoter showing local nucleosomal cond
receptor, a weak agonist at both CB1 and CB2 receptors, and an inhibitor of anandamide reuptake.
y over the closely related 5-HT1D and 5-HT1A receptors.
has some affinity for the 5-HT2A and 5-HT2C receptors, though it is unlikely that these properties
cannabinoid agonist at both the CB1 and CB2 receptors, but with moderate selectivity for CB2, havi
For example, P2X1 and P2X3 receptors desensitize rapidly (a few hundred milliseco
al class that acts on serotonin and dopamine receptors.
e I ( I and IB) and two type II (II and IIB) receptors.
o other areas of the brain, and somatostatin receptors are expressed at many different sites in the
BN acts as a weak agonist of the CB1 and CB2 receptors, with lower affinity in comparison to THC.
be an antagonist at the GABAA and GABAA-rho receptors.
or adrenergic, histaminergic, and muscarinic receptors.
y over the closely related 5-HT2A and 5-HT2C receptors.
s the neurons by activating AMPA and kainate receptors, causing an influx of calcium.
cannabinoid agonist at both the CB1 and CB2 receptors.
albicans as virulence factors and toll-like receptors as relevant mediators of the inflammatory ho
ately equal affinity at both the CB1 and CB2 receptors, having a Ki of 8.0nM at CB1 and 7.0nM at CB
is a potent agonist at both the CB1 and CB2 receptors, with a binding affinity of 0.1nM at CB1 and
Chemokines and their receptors are important for the migration of various c
cannabinoid agonist at both the CB1 and CB2 receptors.
x selectivity over the related Y1, Y2 and Y4 receptors.
s of beta receptor, designated β1, β2 and β3 receptors.
Several ion channels and neurotransmitters receptors pre-mRNa are substrates for ADARs.
Chemokines and their receptors are key regulators of the thymocytes migrati
s in the skin also express NMDA and non-NMDA receptors.
The D1 and D5 receptors are members of the D1-like family of dopamin
ptor antagonist, antagonizing A1, A2, and A3 receptors almost equally, which explains many of its c
Type I and II receptors form a stable complex after ligand binding,
has shown that the nicotinic and muscarinic receptors belong to distinct protein superfamilies.
itionally affinity for the 5-HT2A and 5-HT2C receptors.
It acts on D1 and α receptors as a agonist.
the estrogen, androgen and mineralocorticoid receptors.
ylcholine, GABAA, GABAA-ρ, glycine and 5-HT3 receptors that are composed of five protein subunits w
s found to be an agonist at both CB1 and CB2 receptors.
a ligand for various serotonin and dopamine receptors.
t has moderate affinity for both CB1 and CB2 receptors, with Ki values of 8.36nM at CB1 and 7.95nM
tive for the β1-adrenergic and β2-adrenergic receptors.
cterising the active site of the CB1 and CB2 receptors.
rongest actions are at the 5-HT2B and 5-HT2C receptors and its discriminative cue is mediated prima
interact with muscarinic-2 and muscarinic-4 receptors to inhibit neurally released acetylcholine.
binds to the glucocorticoid and progesterone receptors and therefore blocks the activity of the end
rons may interact with alpha-2A and alpha-2C receptors to inhibit neurally released norepinephrine.
utoreceptor similar to the 5-HT1B and 5-HT1D receptors.
It stimulates dopaminergic and serotonergic receptors and blocks alpha-adrenoreceptors.
tant for neutrophils acting at BLT1 and BLT2 receptors on the plasma membrane of these cells.
entan), which affect both endothelin A and B receptors.
Other neurokinin subtypes and neurokinin receptors that interact with SP have also been reporte
sm is facilitated by D2/D3, nACh, and 5-HT2A receptors.
and has modest affinity for both CB1 and CB2 receptors, with Ki values of 48.3nM at CB1 and 45.5nM
It acts as an agonist at the 5HT2A and 5HT2C receptors.
with the exception of the 5-HT2A and 5-HT2C receptors where it acts as a partial agonist.
cannabinoid agonist at both the CB1 and CB2 receptors.
racts centrally with dopamine and adrenergic receptors.
Warm and cold receptors play a part in sensing innocuous environment
mitragynine acts on the mu- and delta-opiate receptors.
hibitor and agonist at the 5-HT2A and 5-HT2C receptors.
lectivity over the related 5-HT2B and 5-HT2C receptors.
ulation, molecular endocrinology and steroid receptors and transcriptional coactivators.
cannabinoid agonist at both the CB1 and CB2 receptors, with Ki values of 0.46nM at CB1 and 0.69nM
that the differences between GABAС and GABAA receptors are large enough to justify maintaining the
lso partial agonist of µ-opioid and δ-opioid receptors, and competitive antagonist of ϰ-opioid rece
high affinity for the dopamine D2, D3 and D4 receptors, as well as serotonin 5-HT1A and 5-HT2A/C re
ist with high affinity to the D2, D3, and D4 receptors, and with moderate affinity for the 5-HT2 re
y over the closely related 5-HT1D and 5-HT1A receptors.
, and cytoskeletal organization and syndecan receptors are required for internalization of the HIV-
e, suggesting a role for the 5-HT6 and 5-HT7 receptors in regulation of the hearing system.
the antigen receptor (TCR) and costimulatory receptors (CD28 and ICOS) are thought to be main recep
The D1 and D5 receptors have a high degree of structural homology an
selective for Y4 over the related Y1 and Y5 receptors, as the first non-peptide Y4 antagonist deve
Unlike the H1 and H2 receptors which have primarily peripheral actions, but
lectivity over the related 5-HT2A and 5-HT2C receptors.
HT2C), 5-HT6, 5-HT7, α1-adrenergic, and NMDA receptors, and as agonists at the sigma receptors (σ1
activated so that the actions of the δ and κ receptors can be studied separately, in contrast to be
d efficacious agonist at the M1 and δ-opioid receptors, unlike clozapine as well.
s, including stimulation of alpha and beta-1 receptors and inhibition of noradrenaline and tyramine
oximately equal affinity for the CB1 and CB2 receptors, with a Ki of 6.6nM at CB1 and 6.9nM at CB2.
, the ATP sensitivity of P2X1, P2X3 and P2X4 receptors is attenuated when the extracellular pH<7, w
hannels including the GABAA, NMDA, and sigma receptors.
y over the closely related 5-HT2A and 5-HT2C receptors and other receptor targets.
t as non-selective antagonists at A1 and A2A receptors in both heart and brain and so have the oppo
ptor alpha, a subunit shared by IL13 and IL4 receptors.
antagonist at various serotonin and dopamine receptors.
0 mg/day due to its ability to antagonise H3 receptors and thus increase the release of serotonin a
uscle rather than by antagonizing muscarinic receptors.
M, and no significant affinity for any other receptors tested.
eptor" family of apoptosis-inducing cellular receptors.
of its potency, because the appropriate CB1 receptors are not located in brain areas reponsible fo
Several of these are Integrin receptors.
There are numerous receptors that now known to use intracellular kinases
In biochemistry, there are decoy receptors, decoy substrates and decoy RNA.
d to a discovery that there are cell-surface receptors that are stimulated by hormones and neurotra
n potential formation, there are not as many receptors for GABA to bind to - meaning that sympathet
ts like osteoprotegerin as both act as decoy receptors for osteoblastic RANKL.
stranded DNA) molecules which act as surface receptors.
of deep burn there will be no pain as these receptors will be burned off.
As IP3 receptors have sparse or little distribution throughou
clear receptor family such as glucocorticoid receptors.
antiadhesive agents, and second as adhesion receptors.
eins that appear to function as cell-surface receptors for Wnts.
PKC family members also serve as major receptors for phorbol esters, a class of tumor promote
CD16 has been identified as Fc receptors FcγRIIIa and FcγRIIIb.
ked to the D3 activity of pramipexole, as D3 receptors are heavily expressed in brain regions invol
egral proteins that function as cell-surface receptors for Wnts called serpentine receptors.
Hence NCOA2 assists nuclear receptors in the upregulation of DNA expression.
Muscles and associated sensory receptors are innvervated by type I and II sensory fib
rimary tumor mass, and if HA associates with receptors such as CD44, the activation of Rho GTPases
ed to THC, which is a partial agonist at CB1 receptors, JWH-018 (and many of its analogues) are ful
lecule to continuously bind and unbind at β2 receptors in the smooth muscle in the lungs.
acts as a nonselective full agonist at GABAA receptors.
selective, mixed agonist-antagonist at GABAA receptors, which acts as a partial agonist at the α2 a
culline, ROD-188 acts as an agonist at GABAA receptors, being a positive allosteric modulator actin
acts as a partial agonist at benzodiazepine receptors.
ative drug which acts as an agonist at GABAA receptors, specifically acting as a positive allosteri
a subtype-selective partial agonist at GABAA receptors, and was developed by Pfizer as a potential
as, , had a direct action at pharmacological receptors and thereby acting as a neurotransmitter was
shown to possess some activity at serotonin receptors.
n the mammalian brain where it acts at GABAA receptors, which are ligand-gated chloride channels.
the mammalian brain where it acts at GABA-A receptors, which are ligand-gated chloride channels.
Meclozine is an antagonist at H1 receptors.
cts as a partial agonist/antagonist at these receptors.
t acts as an inverse agonist at melanocortin receptors, specifically, MC1.
ts also commonly have action at α-adrenergic receptors and/or 5-HT receptors.
in the mammalian brain where it acts at GABA receptors, which are ligand-gated chloride channels.
e action of aldosterone at mineralocorticoid receptors.
te, some of which also act at benzodiazepine receptors.
the estrogenic potency of coumestrol at both receptors is much less than that of 17β-estradiol.
a subtype-selective partial agonist at GABAA receptors, with highest affinity for the α3 subtype, b
subtype, and little or no affinity at other receptors.
4 is a nonselective partial agonist at GABAA receptors.
nvulsant shown to act as an agonist at GABAA receptors.
the mammalian brain where it acts at GABA-A receptors, which are ligand-gated chloride channels.
The weak agonist activity of pergolide at D1 receptors somewhat alters its clinical and side effect
It is a partial agonist at CB1 receptors, with a Ki of 87nM, making it roughly half t
esearch which acts as an antagonist at GABAB receptors.
t but non-selective partial agonist at GABAA receptors, although with little efficacy at the α1 sub
Semorphone is a partial agonist at μ-opioid receptors.
ion of 2-heptanone as a pheromone at odorant receptors in mice has been investigated.
It is a partial agonist acting at GABAA receptors in the brain.
211, is a mixed agonist-antagonist at opioid receptors with a similar pharmacological profile to pe
phic factor (BDNF), glutamate acting at NMDA receptors, dopamine through activation of the D1 recep
subtype, with little or no affinity at other receptors.
                                                                                                   


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