「EPIDERMAL」の共起表現(1語左で並び替え) - Weblio英語共起表現検索


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Weblio 辞書 > 英和辞典・和英辞典 > EPIDERMALの意味・解説 > EPIDERMALに関連した共起表現

「EPIDERMAL」の共起表現一覧(1語左で並び替え)

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The mine starts as a epidermal corridor, later it becomes a pale and later
ubjects, including discoveries of TGF alpha, epidermal growth factor, and the first to show large-s
The mine starts as an epidermal, whitish or brownish corridor.
The mine starts as an epidermal gallery with a central frass line.
The mine starts as an epidermal corridor, but later becomes a shallow tentif
The mine starts as an epidermal lower-surface corridor that closely follows
The mine starts as an epidermal, sometimes branched, lower-surface corridor,
The mine starts as an epidermal corridor, that later turns into a blotch and
are sensitive to cAMP, AP-1, AP-4, CEBPB and epidermal growth factor.
rease in surface area prevent the dermal and epidermal layers from separating from each others by s
ents, neighboring mesophyll cell layers, and epidermal cells.
and other sensory organs, certain neural and epidermal tissues as well as other homologous structur
luripotent and neural stem cells, as well as epidermal stem cells.
the surface of the skin in hands and feet as epidermal or papillary ridges (colloquially known as f
naling molecules, including mitogens such as epidermal growth factor (EGF), nerve growth factor (NG
s (BMPs); BMPs induce the ectoderm to become epidermal ectoderm.
s a long, lower-surface, strongly branching, epidermal, corridor.
The mine starts as a broad epidermal corridor.
The mine consists of a very long, broad epidermal corridor that winds in dense loops over the
The mine consists of long, broad, epidermal corridor that winds in dense loops over the
eceptor trans-autophosphorylation induced by epidermal growth factor (EGF).
The alpha-neurexins contain epidermal growth factor-like (EGF-like) sequences and
erproliferative and fail to turn on critical epidermal genes.
sation of perspiration; consequent decreased epidermal thermal dissipation leading to warm, blotchy
e growth factor and then went on to discover epidermal growth factor.
"Acantholytic dyskeratotic epidermal nevus" is probably the same disorder.:849
se (also known as "Acantholytic dyskeratotic epidermal nevus") is a cutaneous condition identical t
face of the leaf; at first it is an entirely epidermal and linear gallery, whitish with a glassy lu
me hormones and growth factors, for example, epidermal growth factor and nerve growth factor.
e hybrid structure in which an extracellular epidermal growth factor (EGF)-like domain is fused to
tion by inhibiting fibroblast growth factor, epidermal growth factor, platelet-derived growth facto
y have statocysts which are formed only from epidermal tissue and more than four tentacles and.
ing tiny pale spots or scars where the green epidermal cells have been destroyed.
ptor, VAV2, ADAM15, RAPGEF1, VAV1, HER2/neu, Epidermal growth factor receptor, PDGFRB, PTK2, Erythr
BW 2992, it is a dual inhibitor of the human epidermal growth factor receptor 2 (Her2) and epiderma
bitor (TKI) that irreversibly inhibits human epidermal growth factor receptor 2 (Her2) and epiderma
noclonal antibody directed against the human epidermal growth factor receptor 2 (HER2) with potenti
Expression of functionally active human epidermal growth factor has been done in C. glutamicum
logy, where he has had a special interest in epidermal tumors and Kaposi's sarcoma since 1978.
es and growth factors including interferons, epidermal growth factor, Interleukin 5, Interleukin-6,
Borneo species of snakes that can change its epidermal color spontaneously.
noses include incontinentia pigmenti, linear epidermal nevus, hypomelanosis of Ito and Goltz syndro
The mine consists of a long, epidermal corridor which is located on either the uppe
It starts long-linear, epidermal and running along the leaf vein or leaf marg
Finally, it leaves only upper and lower epidermal layers of the leaf.
The mine starts as a lower-surface epidermal corridor along the midrib.
The mine starts as a lower-surface epidermal gallery that widens into a blotch.
The mine starts with a lower-surface epidermal corridor, but soon the larva starts feeding
The mine starts as a lower-surface epidermal corridor, widening into a blotch, resulting
The mine consists of a lower-surface, epidermal gallery with a very narrow central frass lin
The mine starts as a lower-surface, epidermal corridor, widening into a small full depth b
The larvae start by making a lower-surfsce epidermal corridor.
region with a variable number of N-terminal epidermal growth factor (EGF)-like domains coupled to
extracellular region with several N-terminal epidermal growth factor (EGF)-like domains, which medi
efitinib is the first selective inhibitor of epidermal growth factor receptor's (EGFR) tyrosine kin
It's diagnostic feature is the lack of epidermal invagination at the base of the lophophore.
bring food and oxygen to the lower layers of epidermal cells.
formation of flowers, through the fusion of epidermal cells.
skin and accelerates and improves healing of epidermal wounds.
The larvae feed on the contents of epidermal cells on both top and bottom surfaces of qua
identified by sparse, or dense, coverage of epidermal outgrowths or cirri on the head and body.
found largely in the upper spinous layer of epidermal keratinocytes and mutations in the gene enco
contain a C-type lectin domain, a series of epidermal growth factor like domains (EGF), a highly g
The ErbB protein family or epidermal growth factor receptor (EGFR) family is a fa
hair follicles, sebaceous glands, and other epidermal appendages.
t defines the boundary between proliferative epidermal basal progenitors and terminally differentia
HER2/neu, Wiskott-Aldrich syndrome protein, Epidermal growth factor receptor, PDGFRA, Eukaryotic t
n irregular, oblong, moderate, almost purely epidermal blotch-mine on the upperside of the leaf.
und on the upper side of the leaf and purely epidermal, narrowly linear, very long and tightly coil
mbrane and have nine cadherin domains, seven epidermal growth factor-like repeats and two laminin A
ens into (and is often overrun by) a silvery epidermal upper-surface blotch with light brown frass.
ichuriasis, filariasis,enterobiasis and some epidermal parasitic skin diseases (EPSDs)).
e consists of a long, rather broad, strictly epidermal corridor that curves in dense loops over the
e of ulcerations on the skin, and subsequent epidermal loss, identifiable as white or cloudy fungus
The mine has the form of an upper surface epidermal, silvery blotch without folds.
suggestive of Stevens-Johnson syndrome/toxic epidermal necrolysis.
is a broad term that essentially means that epidermal tissues have become reddened, such as when t
Only the epidermal cells of plants synthesize cutin.
It binds to the epidermal growth factor receptor (EGFR) with high affi
It was originally thought that the epidermal changes were secondary to profound malnutrit
tly induces the vulva fates by secreting the epidermal growth factor (EGF)-like ligand LIN-3.
a appear green (due to chlorophyl) while the epidermal cells appear red due to additional pigmentat
ered by a tunic, which is a secretion of the epidermal cells.
This gene encodes a member of the epidermal growth factor receptor (EGFR) family of rece
ith most fish, as the parasites found on the epidermal layers of the skin and even mouths are one o
ded by this gene binds receptors such as the epidermal growth factor receptor and contains one SH2
elial growth factor receptor (VEGFR) and the epidermal growth factor receptor (EGFR).
of cell surface tyrosine kinases such as the epidermal growth factor receptor.
nds inhibiting the catalytic activity of the epidermal growth factor receptor (EGFR).
EGF acts by binding with high affinity to epidermal growth factor receptor (EGFR) on the cell su
A single case of toxic epidermal necrolysis was reported in 1968.
1996, due to rare but serious cases of toxic epidermal necrolysis.
yndrome, Stevens-Johnson syndrome, and toxic epidermal necrolysis (SJS/TEN).
ic shock, Stevens-Johnson syndrome and toxic epidermal necrolysis (TEN).
oral ciprofloxacin administration and toxic epidermal necrolysis (TEN) or Stevens-Johnson syndrome
of cases of Stevens-Johnson syndrome, toxic epidermal necrolysis and erythema multiforme, as well
The miracidia is composed of twenty epidermal plates arranged in 4 tiers: 6, 8, 4, 2 cells
Mines are always started at the upper epidermal layer.
The mine at first is narrowly linear, upper epidermal, and transparent-whitish in colour, then it
They create a silvery, upper-surface, epidermal tentiform mine, which is centred over the mi
mine consists of a lower- or upper-surface, epidermal corridor that usually begins at the midrib a
, lower-surface (but sometimes upper-surfce) epidermal, silvery corridor.
he Walleye dermal sarcoma virus, and Walleye epidermal hyperplasia virus 1 and 2.
bes with a bumpy textured surface and a waxy epidermal coating.
The mine consists of a winding, epidermal corridor, resembling a snail's trail, runnin
AS/BRAF (which are oncogenes associated with epidermal growth factor receptor EGFR), RNA stabilizat
                                                                                                   


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