「EPIDERMAL」の共起表現(2語左で並び替え)

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共起表現

「EPIDERMAL」の共起表現一覧(2語左で並び替え)

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The mine starts as a broad	epidermal corridor.
The larvae start by making a lower-surfsce	epidermal corridor.
The mine starts as a lower-surface	epidermal corridor along the midrib.
The mine starts as a lower-surface	epidermal gallery that widens into a blotch.
The mine consists of a lower-surface,	epidermal gallery with a very narrow central frass lin
bes with a bumpy textured surface and a waxy	epidermal coating.
The mine starts with a lower-surface	epidermal corridor, but soon the larva starts feeding
The mine consists of a winding,	epidermal corridor, resembling a snail's trail, runnin
The mine starts as a lower-surface,	epidermal corridor, widening into a small full depth b
The mine consists of a long,	epidermal corridor which is located on either the uppe
The mine starts as a lower-surface	epidermal corridor, widening into a blotch, resulting
ens into (and is often overrun by) a silvery	epidermal upper-surface blotch with light brown frass.
Expression of functionally active human	epidermal growth factor has been done in C. glutamicum
EGF acts by binding with high affinity to	epidermal growth factor receptor (EGFR) on the cell su
n irregular, oblong, moderate, almost purely	epidermal blotch-mine on the upperside of the leaf.
The alpha-neurexins contain	epidermal growth factor-like (EGF-like) sequences and
e hybrid structure in which an extracellular	epidermal growth factor (EGF)-like domain is fused to
face of the leaf; at first it is an entirely	epidermal and linear gallery, whitish with a glassy lu
Finally, it leaves only upper and lower	epidermal layers of the leaf.
he Walleye dermal sarcoma virus, and Walleye	epidermal hyperplasia virus 1 and 2.
hair follicles, sebaceous glands, and other	epidermal appendages.
e of ulcerations on the skin, and subsequent	epidermal loss, identifiable as white or cloudy fungus
und on the upper side of the leaf and purely	epidermal, narrowly linear, very long and tightly coil
elial growth factor receptor (VEGFR) and the	epidermal growth factor receptor (EGFR).
yndrome, Stevens-Johnson syndrome, and toxic	epidermal necrolysis (SJS/TEN).
ic shock, Stevens-Johnson syndrome and toxic	epidermal necrolysis (TEN).
oral ciprofloxacin administration and toxic	epidermal necrolysis (TEN) or Stevens-Johnson syndrome
ichuriasis, filariasis,enterobiasis and some	epidermal parasitic skin diseases (EPSDs)).
The mine starts as an	epidermal, whitish or brownish corridor.
The mine starts as an	epidermal gallery with a central frass line.
The mine starts as an	epidermal corridor, but later becomes a shallow tentif
The mine starts as an	epidermal lower-surface corridor that closely follows
The mine starts as an	epidermal, sometimes branched, lower-surface corridor,
ded by this gene binds receptors such as the	epidermal growth factor receptor and contains one SH2
The mine starts as a	epidermal corridor, later it becomes a pale and later
of cell surface tyrosine kinases such as the	epidermal growth factor receptor.
The mine starts as an	epidermal corridor, that later turns into a blotch and
AS/BRAF (which are oncogenes associated with	epidermal growth factor receptor EGFR), RNA stabilizat
t defines the boundary between proliferative	epidermal basal progenitors and terminally differentia
e consists of a long, rather broad, strictly	epidermal corridor that curves in dense loops over the
are sensitive to cAMP, AP-1, AP-4, CEBPB and	epidermal growth factor.
Borneo species of snakes that can change its	epidermal color spontaneously.
sation of perspiration; consequent decreased	epidermal thermal dissipation leading to warm, blotchy
The larvae feed on the contents of	epidermal cells on both top and bottom surfaces of qua
identified by sparse, or dense, coverage of	epidermal outgrowths or cirri on the head and body.
rease in surface area prevent the dermal and	epidermal layers from separating from each others by s
mbrane and have nine cadherin domains, seven	epidermal growth factor-like repeats and two laminin A
The ErbB protein family or	epidermal growth factor receptor (EGFR) family is a fa
the surface of the skin in hands and feet as	epidermal or papillary ridges (colloquially known as f
me hormones and growth factors, for example,	epidermal growth factor and nerve growth factor.
formation of flowers, through the fusion of	epidermal cells.
tion by inhibiting fibroblast growth factor,	epidermal growth factor, platelet-derived growth facto
skin and accelerates and improves healing of	epidermal wounds.
es and growth factors including interferons,	epidermal growth factor, Interleukin 5, Interleukin-6,
eceptor trans-autophosphorylation induced by	epidermal growth factor (EGF).
efitinib is the first selective inhibitor of	epidermal growth factor receptor's (EGFR) tyrosine kin
bitor (TKI) that irreversibly inhibits human	epidermal growth factor receptor 2 (Her2) and epiderma
logy, where he has had a special interest in	epidermal tumors and Kaposi's sarcoma since 1978.
It's diagnostic feature is the lack of	epidermal invagination at the base of the lophophore.
found largely in the upper spinous layer of	epidermal keratinocytes and mutations in the gene enco
bring food and oxygen to the lower layers of	epidermal cells.
ents, neighboring mesophyll cell layers, and	epidermal cells.
The mine at first is narrowly linear, upper	epidermal, and transparent-whitish in colour, then it
The mine consists of long, broad,	epidermal corridor that winds in dense loops over the
The mine consists of a very long, broad	epidermal corridor that winds in dense loops over the
is a broad term that essentially means that	epidermal tissues have become reddened, such as when t
(BAF), leukocyte endogenous mediator (LEM),	epidermal cell-derived thymocyte-activating factor (ET
and other sensory organs, certain neural and	epidermal tissues as well as other homologous structur
A single case of toxic	epidermal necrolysis was reported in 1968.
1996, due to rare but serious cases of toxic	epidermal necrolysis.
ered by a tunic, which is a secretion of the	epidermal cells.
This gene encodes a member of the	epidermal growth factor receptor (EGFR) family of rece
The miracidia is composed of twenty	epidermal plates arranged in 4 tiers: 6, 8, 4, 2 cells
nds inhibiting the catalytic activity of the	epidermal growth factor receptor (EGFR).
region with a variable number of N-terminal	epidermal growth factor (EGF)-like domains coupled to
erproliferative and fail to turn on critical	epidermal genes.
ith most fish, as the parasites found on the	epidermal layers of the skin and even mouths are one o
Only the	epidermal cells of plants synthesize cutin.
y have statocysts which are formed only from	epidermal tissue and more than four tentacles and.
mine consists of a lower- or upper-surface,	epidermal corridor that usually begins at the midrib a
noses include incontinentia pigmenti, linear	epidermal nevus, hypomelanosis of Ito and Goltz syndro
tly induces the vulva fates by secreting the	epidermal growth factor (EGF)-like ligand LIN-3.
contain a C-type lectin domain, a series of	epidermal growth factor like domains (EGF), a highly g
extracellular region with several N-terminal	epidermal growth factor (EGF)-like domains, which medi
They create a silvery, upper-surface,	epidermal tentiform mine, which is centred over the mi
, lower-surface (but sometimes upper-surfce)	epidermal, silvery corridor.
It starts long-linear,	epidermal and running along the leaf vein or leaf marg
suggestive of Stevens-Johnson syndrome/toxic	epidermal necrolysis.
s a long, lower-surface, strongly branching,	epidermal, corridor.
naling molecules, including mitogens such as	epidermal growth factor (EGF), nerve growth factor (NG
Stevens-Johnson syndrome (toxic	epidermal necrolysis) when mebendazole is combined wit
HER2/neu, Wiskott-Aldrich syndrome protein,	Epidermal growth factor receptor, PDGFRA, Eukaryotic t
of cases of Stevens-Johnson syndrome, toxic	epidermal necrolysis and erythema multiforme, as well
ubjects, including discoveries of TGF alpha,	epidermal growth factor, and the first to show large-s
It was originally thought that the	epidermal changes were secondary to profound malnutrit
Mines are always started at the upper	epidermal layer.
BW 2992, it is a dual inhibitor of the human	epidermal growth factor receptor 2 (Her2) and epiderma
noclonal antibody directed against the human	epidermal growth factor receptor 2 (HER2) with potenti
ing tiny pale spots or scars where the green	epidermal cells have been destroyed.
It binds to the	epidermal growth factor receptor (EGFR) with high affi
s (BMPs); BMPs induce the ectoderm to become	epidermal ectoderm.
e growth factor and then went on to discover	epidermal growth factor.
The mine has the form of an upper surface	epidermal, silvery blotch without folds.
ptor, VAV2, ADAM15, RAPGEF1, VAV1, HER2/neu,	Epidermal growth factor receptor, PDGFRB, PTK2, Erythr
luripotent and neural stem cells, as well as	epidermal stem cells.
a appear green (due to chlorophyl) while the	epidermal cells appear red due to additional pigmentat

共起表現

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「EPIDERMAL」の共起表現一覧(2語左で並び替え)