「ca 2」の共起表現一覧(1語右で並び替え)
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TsTx-IV blocks | Ca2+ activated K+ channels of high conductance. |
-Ca2+ activated K+ Channel affecting peptides: TsTX- | |
ied by compound screening to be an activator of | Ca2+ activated Cl- channels (CaCCs), thus a potentia |
potassium channels: the voltage-activated, the | Ca2+- activated and the inward-rectifier potassium c |
Its hormone binds to a receptor, and | Ca2+ activates a protein, calmodulin, and the comple |
This protein has a | Ca2+ affinity 20- to 100-fold higher than the other |
Hard water has high concentrations of | Ca2+ and Mg2+ ions. |
The hormone participates in calcium ( | Ca2+) and phosphorus metabolism. |
most important ions in the 5th group are Ba2+, | Ca2+, and Sr2+. |
release (exocytosis) is dependent upon calcium | Ca2+ and is a presynaptic response. |
ranes in response to physiological increases in | Ca2+ and selectively hydrolyses arachidonyl phosphol |
the annexin family of evolutionarily conserved | Ca2+ and phospholipid binding proteins. |
Furthermore, the KCa1 family is both | Ca2+ and voltage activated, further complicating the |
, for the most part, activated by intracellular | Ca2+ and contains 8 members. |
inorganic salts (mostly Na+, Cl-, K+, Mg2+, and | Ca2+), and organic compounds (mostly carbohydrates, |
IPA interaction with the BK channel enhances | Ca2+ and / or voltage sensitivity of the α subunit o |
t known formation constant for the complexation | Ca2+ and Gd3+ ions. |
tivation of TRPC6 induces the entry of calcium ( | Ca2+) and sodium (Na+) into the cell, which results |
e humite series Mg2+ is replaced by Fe2+, Mn2+, | Ca2+ and Zn2+ in that order of abundance, though Mg2 |
selective for Mn2+, somewhat less selective for | Ca2+ and Mg2+, much less selective for Sr2+, and eve |
with divalent metallic cations such as calcium ( | Ca2+) and iron(II) (Fe2+) to form crystals of the co |
ished: the stoichiometry of exchange is 3 Na+:1 | Ca2+ and the exchanger is electrogenic and voltage-s |
Chelators for | Ca2+ are well established, have high affinity for th |
of the calcium, and ionic solutions of calcium ( | Ca2+) are colorless as well. |
ex is made up of tissue factor, factor VII, and | Ca2+ as an activating ion. |
lar biochemistry of granins includes binding of | Ca2+, ATP and catecholamines (epinephrine, norepinep |
Plasma membrane | Ca2+ ATPase (PMCA) |
Sarcoplasmic reticulum | Ca2+ ATPase (SERCA) |
The plasma membrane | Ca2+ ATPase (PMCA) is a transport protein in the pla |
which stands for sarco / endoplasmic reticulum | Ca2+ ATPase. |
Beauvericin ( | Ca2+, Ba2+) |
of Ca2+-mediated events occur when the released | Ca2+ binds to and activates the regulatory protein c |
k to this surface through their Gla domain with | Ca2+ bridges. |
(R3, R4), Rodalies Barcelona line R7, Ca1, | Ca2, Ca3, Ca4, Ca6, R42) |
Ca2+, calcium is a component of bones and teeth. | |
Constitutively high levels of mitochondrial | Ca2+ cause inappropriate MPTP opening leading to a d |
ashout could only moderately reverse the R-type | Ca2+ channel inhibition after treatment with 200 nM |
is stimulatory to adenylyl cyclase, acts on the | Ca2+ channel directly as an effector. |
R) is a membrane glycoprotein complex acting as | Ca2+ channel activated by inositol trisphosphate (In |
fer into rabbit erythrocytes (red blood cells), | Ca2+ channel antagonistic action, α1 adrenergic bloc |
ha1E, R-type) channel (strong affinity), L-type | Ca2+ channel, P/Q type Ca2+ channel, Na+ channel . |
asmic reticulum and mitochondria, help open the | Ca2+ channel. |
s direct gating has also been found in specific | Ca2+ channels in the heart and skeletal muscle T tub |
own that it can also inhibit L-type or P/Q type | Ca2+ channels and incompletely block Na+ channels. |
g mobilization of calcium ions through specific | Ca2+ channels into the cytosol. |
es and associated sarcolemmal vesicles in which | Ca2+ channels are able to survive and function in th |
n cAMP in the cell, inhibition of voltage-gated | Ca2+ channels, and efflux of K+, in general, leading |
strong voltage reverses the blocking of R-type | Ca2+ channels. |
Fluo-4 is used to measure calcium ( | Ca2+) concentrations inside living cells, and is oft |
cation is primarily controlled by the change in | Ca2+ concentrations, causing excitability within the |
e together the main regulators of intracellular | Ca2+ concentrations. |
leased is referred to as Ca2+-release-activated | Ca2+ current (ICRAC). |
SNX-482 inhibits native R-type | Ca2+ currents at weak nanomolar concentrations in ra |
However, it does not influence R-type | Ca2+ currents at concentrations of 200-500 nM in sev |
If a cell has Na+ or | Ca2+ currents at rest, then inhibition of those curr |
synaptotagmin, among others, in the presence of | Ca2+, displaces complexin allowing the SNARE protein |
large transmembrane electrochemical gradient of | Ca2+ driving the entry of the ion into cells, yet it |
ium from the endoplasmic reticulum will lead to | Ca2+ entry from outside the cell by activation of "S |
l loop, diuretics cause an increase in Mg2+ and | Ca2+ excretion. |
vated by voltage, while others are activated by | Ca2+, extracellular ligands, and pH among other modu |
he addition of another required factor, such as | Ca2+ for the photoprotein aequorin. |
tually it contains only divalent ions, Mg2+ and | Ca2+ for cation exchange resins, and SO42- for anion |
this is because of a biological requirement for | Ca2+ for the protein to fold into the correct form. |
In fact, the PMCA is involved in removing | Ca2+ from all eukaryotic cells. |
nant second messenger leading to the release of | Ca2+ from intracellular store sites. |
gradient generated by the Na+-K+ pump to remove | Ca2+ from the intracellular space, slowing down the |
ology, two photon laser scanning microscopy and | Ca2+ imaging have been used to study activity at the |
ditionally, honokiol increases free cytoplasmic | Ca2+ in rat cortical neurons. |
The | Ca2+ in turn activates chloride channels, causing ef |
It increases the level of calcium ( | Ca2+) in the blood by (1) increasing the uptake of c |
IP3 is one of the most effective inducer of | Ca2+ increase from cytoplasmic pools and from outsid |
bited by Rhoa and Rho kinase, components of the | Ca2+ independent pathway for maintaining muscle cont |
gonists reversibly block NMDA receptor-mediated | Ca2+ influx and thus may inhibit excitotoxicity. |
nerves to their terminals where they initiate a | Ca2+ influx and the release of acetylcholine (ACh). |
Since it transports | Ca2+ into the extracellular space, the PMCA is also |
y a key role in the modulation of intracellular | Ca2+ involved in presynaptic neurotransmitter releas |
leads to the closure of cGMP-regulated Na+ and | Ca2+ ion channels and a hyperpolarized membrane pote |
ginate forms an adhesive gel in the presence of | Ca2+ ion. |
Ca2+ ions are a key component to muscle contraction. | |
The increase in intracellular | Ca2+ ions induces the RYR to release even more Ca2+ |
is opening allows for an influx of both Na+ and | Ca2+ ions into the cell, thus depolarizing it. |
channels are activated and cause the influx of | Ca2+ ions over the membrane and to the release of ca |
lectroneutrality requires that the transport of | Ca2+ ions catalyzed by the intestinal epithelial cel |
Ca2+ ions can damage cells if they enter in excessiv | |
nlike most TRP channels, TRPV6 is selective for | Ca2+ ions, similarly to its close homologue, TRPV5, |
nges of electric field, pH, or concentration of | Ca2+ ions. |
inlet for the action potential near a source of | Ca2+ ions. |
(6290) 1985 | CA2 is a main-belt minor planet. |
For GnRH, TRH and GHRH the increase in | Ca2+ is achieved by the releasing hormone coupling a |
Since | Ca2+ is actively reabsorbed in the distal convoluted |
Calcium ( | Ca2+) is often considered part of the BMP, though, b |
The effects of | Ca2+ is also remarkable: it cooperates with DAG in a |
is dye to cell types where the resting level of | Ca2+ is < 1 μM and does not vary with the experiment |
d cations, to cation selective allowing passage | Ca2+, K+ and Na+, a highly selective K+ channels. |
used in laboratories to increase intracellular | Ca2+ levels in intact cells. |
result of the process is increased cytoplasmic | Ca2+ levels via the direct pathway described above a |
higher concentrations of divalent ions (SO42-, | Ca2+, Mg2+) and lower Na+ and Cl- than the surroundi |
obscures the part of the molecule that chelates | Ca2+, Mg2+, Zn2+ and other ions. |
ls, which play critical roles in proliferation, | Ca2+ mobilization and cell differentiation. |
It is involved in B-cell receptor induced | Ca2+ mobilization from intracellular stores and prom |
in part through the regulation of intracellular | Ca2+ mobilization. |
OX2 receptor activation, such as intracellular | Ca2+ mobilization. |
t each of the 3 positions have an impact on the | Ca2+ permeability of the channel |
Rendered image of the | Ca2+ pump |
connections to R1 and R2 Rodalies trains and to | Ca2 regional trains. |
he sarcoplasmic reticulum may cause spontaneous | Ca2+ release during repolarization, causing the rele |
se) the intracellular concentration of calcium ( | Ca2+), resulting in vesicle fusion of the respective |
by synaptobrevin, syntaxin and SNAP-25) and the | Ca2+ sensor synaptotagmin. |
ell as increasing the overall complexity of the | Ca2+ signaling mechanism. |
conversion of external stimuli to intracellular | Ca2+ signals characterized by complex patterns relat |
and affect the flow of potassium (K+), calcium ( | Ca2+), sodium (Na+), and chloride (Cl-) across the p |
c inhibitor of Ca2+-ATPase in the intracellular | Ca2+ storage sites. |
he positions of the ions are reversed: calcium ( | Ca2+) take the oxide (O2−) positions and nitride ion |
nts for the sustained transport of ions such as | Ca2+ that controls T lymphocyte (T cell) proliferati |
Na+ through Na+ channels, or | Ca2+ through Ca2+ channels. |
Na+ through Na+ channels or | Ca2+ through Ca2+ channels, inhibits hyperpolarizati |
r disease state by increasing the intracellular | Ca2+ to increase the contractility cycling rates. |
ns as a positive regulator of the transcellular | Ca2+ transport pathway, and it plays a role in the i |
For example, | Ca2+ waves and oscillations. |
It is vital for regulating the amount of | Ca2+ within cells. |
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